The Salmonella Pathogenicity Island (SPI)-2 and SPI-1 type III secretion systems allow Salmonella serovar typhimurium to trigger colitis via MyD88-dependent and MyD88-independent mechanisms

Journal of Immunology

Volumn 174, Issue 3, 2005, Pages 1675-1685

  Hapfelmeier, Siegfried ^a   Stecher, Bärbel ^a   Barthel, Manja ^a   Kremer, Marcus ^d   Müller, Andreas J ^a   Heikenwalder, Mathias ^b   Stallmach, Thomas ^c   Hensel, Michael ^b   Pfeffer, Klaus ^e   Akira, Shizuo ^f,g   Hardt, Wolf Dietrich ^a  

^a ETH ZURICH   (Switzerland)

^b UNIVERSITY HOSPITAL ERLANGEN   (Germany)

^c UNIVERSITY HOSPITAL ZURICH   (Switzerland)

^d TECHNICAL UNIVERSITY OF MUNICH   (Germany)

^e HEINRICH HEINE UNIVERSITY   (Germany)

^f OSAKA UNIVERSITY   (Japan)

^g Japan Science and Technology Agency   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

BACTERIAL PROTEIN; CD11 ANTIGEN; MYELOID DIFFERENTIATION FACTOR 88; STREPTOMYCIN; TOLL LIKE RECEPTOR;

ANIMAL CELL; ARTICLE; CELL DISRUPTION; CELL GROWTH; CELL POPULATION; CELL PROLIFERATION; COLITIS; IMMUNE RESPONSE; IMMUNITY; IN VITRO STUDY; IN VIVO STUDY; INFLAMMATION; INTESTINE EPITHELIUM; KNOCKOUT MOUSE; LAMINA PROPRIA; LEUKOCYTE; MOUSE; MUCOSA INFLAMMATION; MUTANT; NONHUMAN; PATHOGENICITY ISLAND; PRIORITY JOURNAL; SALMONELLA; SALMONELLA TYPHIMURIUM; SEROTYPE; WILD TYPE;

EID: 19944430409     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.174.3.1675     Document Type: Article

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