Opening of Ca2+-activated K+ channels triggers early and delayed preconditioning against I/R injury independent of NOS in mice

American Journal of Physiology - Heart and Circulatory Physiology

Volumn 287, Issue 5 56-5, 2004, Pages

  Wang, Xiaoyin ^a   Yin, Chang ^a   Xi, Lei ^a   Kukreja, Rakesh C ^a,b  

^a VETERANS AFFAIRS MEDICAL CENTER   (United States)

^b VIRGINIA COMMONWEALTH UNIVERSITY   (United States)

Author keywords

Ischemia reperfusion; Nitric oxide synthase; NS 1619; Pharmacological preconditioning; Signaling

Indexed keywords

1 (2 HYDROXY 5 TRIFLUOROMETHYLPHENYL) 5 TRIFLUOROMETHYL 2(3H) BENZIMIDAZOLONE; ADENOSINE TRIPHOSPHATE; CALCIUM ACTIVATED POTASSIUM CHANNEL; DIAZOXIDE; DIMETHYL SULFOXIDE; DRUG VEHICLE; ENDOTHELIAL NITRIC OXIDE SYNTHASE; INDUCIBLE NITRIC OXIDE SYNTHASE; N(G) NITROARGININE METHYL ESTER; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE INHIBITOR; PAXILLINE; POTASSIUM CHANNEL; POTASSIUM CHANNEL BLOCKING AGENT; BENZIMIDAZOLE DERIVATIVE; CARDIOTONIC AGENT; ISOPROTEIN; NOS2 PROTEIN, MOUSE; NOS3 PROTEIN, MOUSE;

ALPHA CHAIN; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BETA CHAIN; CONTROLLED STUDY; CONVALESCENCE; DRUG EFFECT; DRUG INFUSION; HEART; HEART INFARCTION; HEART MUSCLE ISCHEMIA; HEART PROTECTION; HEART VENTRICLE FUNCTION; MALE; MITOCHONDRION; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; REPERFUSION INJURY; TIME; WESTERN BLOTTING; ANIMAL; HEART INFARCTION PREVENTION; METABOLISM; METHODOLOGY; OUTBRED STRAIN; PATHOLOGY; PATHOPHYSIOLOGY;

ANIMALS; ANIMALS, OUTBRED STRAINS; BENZIMIDAZOLES; CARDIOTONIC AGENTS; ISCHEMIC PRECONDITIONING, MYOCARDIAL; MALE; MICE; MYOCARDIAL INFARCTION; MYOCARDIAL ISCHEMIA; MYOCARDIAL REPERFUSION INJURY; NITRIC OXIDE SYNTHASE; NITRIC OXIDE SYNTHASE TYPE II; NITRIC OXIDE SYNTHASE TYPE III; POTASSIUM CHANNELS, CALCIUM-ACTIVATED; PROTEIN ISOFORMS; TIME FACTORS; VENTRICULAR FUNCTION;

EID: 16544378948     PISSN: 03636135     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.00431.2004     Document Type: Article

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