Inhibition of endogenous thioredoxin in the heart increases oxidative stress and cardiac hypertrophy

Journal of Clinical Investigation

Volumn 112, Issue 9, 2003, Pages 1395-1406

  Yamamoto, Mitsutaka ^a   Yang, Guiping ^a   Hong, Chull ^a   Liu, Jing ^a   Holle, Eric ^b   Yu, Xianzhong ^b   Wagner, Thomas ^b   Vatner, Stephen F ^a   Sadoshima, Junichi ^a  

^a RUTGERS NEW JERSEY MEDICAL SCHOOL   (United States)

^b Oncology Research Institute   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIOXIDANT; CYSTEINE; MITOGEN ACTIVATED PROTEIN KINASE; RAF PROTEIN; RAS PROTEIN; THIOREDOXIN; TIOPRONIN;

ANIMAL MODEL; ANIMAL TISSUE; ANTIOXIDANT ACTIVITY; ARTICLE; CONTROLLED STUDY; DOMINANT INHERITANCE; DOMINANT NEGATIVE MUTANT; ECHOCARDIOGRAPHY; GENE OVEREXPRESSION; HEART FUNCTION; HEART HYPERTROPHY; HEART PRESSURE OVERLOAD; HEART VENTRICLE OVERLOAD; MOUSE; MUTANT; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; TRANSGENIC MOUSE;

ANIMALS; CARDIOMEGALY; CATALASE; CELLS, CULTURED; MICE; MICE, TRANSGENIC; MITOGEN-ACTIVATED PROTEIN KINASES; OXIDATIVE STRESS; P38 MITOGEN-ACTIVATED PROTEIN KINASES; PROTO-ONCOGENE PROTEINS C-RAF; SUPEROXIDE DISMUTASE; THIOREDOXIN;

EID: 0347986778     PISSN: 00219738     EISSN: None     Source Type: Journal    
DOI: 10.1172/JCI200317700     Document Type: Article

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