ERK and p38 mediate high-glucose-induced hypertrophy and TGF-β expression in renal tubular cells

American Journal of Physiology - Renal Physiology

Volumn 286, Issue 1 55-1, 2004, Pages

  Fujita, Hisayo ^a   Omori, Sayu ^a   Ishikura, Kenji ^a   Hida, Mariko ^a   Awazu, Midori ^a  

^a KEIO UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

Diabetes; Kidney; Mitogen activated protein kinase

Indexed keywords

2 (2 AMINO 3 METHOXYPHENYL)CHROMONE; 4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; DNA; GLUCOSE; LEUCINE; MITOGEN ACTIVATED PROTEIN KINASE; MITOGEN ACTIVATED PROTEIN KINASE INHIBITOR; MITOGEN ACTIVATED PROTEIN KINASE P38; PROTEIN; STREPTOZOCIN; TRANSFORMING GROWTH FACTOR BETA; TRITIUM;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; CELL CULTURE; CELL LINE; CONTROLLED STUDY; CULTURE MEDIUM; DIABETES MELLITUS; DISEASE MODEL; ENZYME ACTIVATION; HYPERTROPHY; KIDNEY TUBULE CELL; MALE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN LOCALIZATION; RAT; RAT STRAIN; STREPTOZOCIN DIABETES;

EID: 0347360288     PISSN: 1931857X     EISSN: 15221466     Source Type: Journal    
DOI: 10.1152/ajprenal.00351.2002     Document Type: Article

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