L-type Ca2+ channels provide a major pathway for iron entry into cardiomyocytes in iron-overload cardiomyopathy

Nature Medicine

Volumn 9, Issue 9, 2003, Pages 1187-1194

  Oudit, Gavin Y ^c   Sun, Hui ^d   Trivieri, Maria G ^d   Koch, Sheryl E ^b   Dawood, Fayez ^d   Ackerley, Cameron ^a   Yazdanpanah, Mehrdad ^d   Wilson, Greg J ^a   Schwartz, Arnold ^b   Liu, Peter P ^c   Backx, Peter H ^c  

^a HOSPITAL FOR SICK CHILDREN   (Canada)

^b UNIVERSITY OF CINCINNATI MEDICAL CENTER   (United States)

^c UNIVERSITY HEALTH NETWORK   (Canada)

^d NONE

Author keywords

[No Author keywords available]

Indexed keywords

AMLODIPINE; CALCIUM CHANNEL BLOCKING AGENT; CALCIUM CHANNEL L TYPE; VERAPAMIL; CALCIUM CHANNEL; IRON;

ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRADYCARDIA; CARDIOMYOPATHY; CONTROLLED STUDY; DIASTOLIC BLOOD PRESSURE; DRUG EFFECT; GENE OVEREXPRESSION; HEART FUNCTION; HEART MUSCLE CELL; HEART MUSCLE FIBROSIS; HYPOTENSION; IRON OVERLOAD; IRON TRANSPORT; MALE; MORTALITY; MOUSE; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; PROGNOSIS; SURVIVAL; SYSTOLIC BLOOD PRESSURE; TRANSGENIC MOUSE; ANIMAL; HEART MUSCLE; METABOLISM; MOUSE STRAIN; PATHOLOGY; SURVIVAL RATE; TRANSPORT AT THE CELLULAR LEVEL;

ANIMALIA; MUS MUSCULUS;

AMLODIPINE; ANIMALS; BIOLOGICAL TRANSPORT; CALCIUM CHANNEL BLOCKERS; CALCIUM CHANNELS; CARDIOMYOPATHIES; IRON; IRON OVERLOAD; MALE; MICE; MICE, INBRED STRAINS; MYOCARDIUM; MYOCYTES, CARDIAC; OXIDATIVE STRESS; SURVIVAL RATE; VERAPAMIL;

EID: 0141461407     PISSN: 10788956     EISSN: None     Source Type: Journal    
DOI: 10.1038/nm920     Document Type: Article

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