Ca2+ signaling in cardiac myocytes overexpressing the α1 subunit of L-type Ca2+ channel

Circulation Research

Volumn 90, Issue 2, 2002, Pages 174-181

  Song, Long Sheng ^a   Guia, António ^a   Muth, James N ^b   Rubio, Marta ^b   Wang, Shi Qiang ^a   Xiao, Rui Ping ^a   Josephson, Ira R ^a   Lakatta, Edward G ^a   Schwartz, Arnold ^b   Cheng, Heping ^a  

^a NATIONAL INSTITUTE ON AGING   (United States)

^b UNIVERSITY OF CINCINNATI   (United States)

Author keywords

Ca2+ homeostasis; Ca2+ sparks; Cardiac hypertrophy; Excitation contraction coupling; L type Ca2+ channels

Indexed keywords

CALCIUM; CALCIUM CHANNEL L TYPE; PHOSPHOLAMBAN; RYANODINE RECEPTOR;

ANIMAL CELL; ARTICLE; CALCIUM CELL LEVEL; CALCIUM HOMEOSTASIS; CALCIUM SIGNALING; CALCIUM TRANSPORT; CELL DENSITY; CHANNEL GATING; CONTROLLED STUDY; EXCITATION CONTRACTION COUPLING; GENE INACTIVATION; GENE OVEREXPRESSION; HEART MUSCLE CELL; HEART VENTRICLE HYPERTROPHY; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SODIUM CALCIUM EXCHANGE; TRANSGENIC MOUSE; TRANSPORT KINETICS; WESTERN BLOTTING;

ACTION POTENTIALS; ANIMALS; CAFFEINE; CALCIUM; CALCIUM CHANNELS, L-TYPE; CALCIUM SIGNALING; CALCIUM-BINDING PROTEINS; CALCIUM-TRANSPORTING ATPASES; CARDIAC PACING, ARTIFICIAL; CARDIOMEGALY; CELL SEPARATION; CYTOSOL; HOMEOSTASIS; MICE; MICE, TRANSGENIC; MYOCARDIUM; PATCH-CLAMP TECHNIQUES; PROTEIN SUBUNITS; RYANODINE RECEPTOR CALCIUM RELEASE CHANNEL; SARCOLEMMA; SARCOPLASMIC RETICULUM CALCIUM-TRANSPORTING ATPASES; SODIUM-CALCIUM EXCHANGER;

EID: 0037040149     PISSN: 00097330     EISSN: None     Source Type: Journal    
DOI: 10.1161/hh0202.103230     Document Type: Article

References (31)

1. Generation, control, and processing of cellular calcium signals
2. 2+ channels
3. Time and calcium dependence of activation and inactivation of calcium-induced release of calcium from the sarcoplasmic reticulum of a skinned canine cardiac Purkinje cell
4. 2+ exchanger
5. Isolation of phospholamban and a second proteolipid component from canine cardiac sarcoplasmic reticulum
6. Calcium sparks and excitation-contraction coupling in phospholamban-deficient mouse ventricular myocytes
7. 2+ signaling in transgenic mouse cardiac myocytes overexpressing calsequestrin
8. Calcium sparks: Elementary events underlying excitation-contraction coupling in heart muscle
9. 2+ channels and ryanodine receptors in heart cells
10. Calcium fluxes involved in control of cardiac myocyte contraction
11. Local calcium transients triggered by single L-type calcium channel currents in cardiac cells
12. The control of calcium release in heart muscle
13. 2+ sparks and local control theories for cardiac excitation-contraction coupling
14. 2+-dependent transgenic model of cardiac hypertrophy: A role for protein kinase Cα
15. Cellular mechanisms of altered contractility in the hypertrophied heart: Big hearts, big sparks
16. Cell calcium, oncogenes, and hypertrophy
17. Abnormal biochemistry in myocardial failure
18. ic subunit
19. 2+ channel in transgenic mice: Loss of isoproterenol-induced contraction
20. Culture and adenoviral infection of adult mouse cardiac myocytes: Methods for cellular genetic physiology
21. Ion concentration-dependence of rat cardiac unitary L-type calcium channel conductance
22. Amplitude distribution of calcium sparks in confocal images: Theory and studies with an automatic detection method
23. Local control of excitation-contraction coupling in rat heart cells
24. 2+ release during excitation-contraction coupling in cardiac myocytes
25. Local control models of cardiac excitation-contraction coupling: A possible role for allosteric interactions between ryanodine receptors
26. 2+ current in single rat ventricular cells
27. Defective excitation-contraction coupling in experimental cardiac hypertrophy and heart failure
28. 2+ signaling
29. 2+ channels and ryanodine receptors in rat ventricular myocytes
30. 2+ spikes' in rat cardiac myocytes
31. Geometric parameters of pipettes and membrane patches