Fas-stimulated generation of reactive oxygen species or exogenous oxidative stress sensitize cells to Fas-mediated apoptosis

Free Radical Biology and Medicine

Volumn 35, Issue 6, 2003, Pages 648-661

  Devadas, Satish ^a   Hinshaw, Jennifer A ^a,b   Zaritskaya, Liubov ^a   Williams, Mark S ^a,b,c  

^a AMERICAN RED CROSS   (United States)

^b GEORGE WASHINGTON UNIVERSITY SCHOOL OF MEDICINE AND HEALTH SCIENCES   (United States)

^c HOLLAND LABORATORY   (United States)

Author keywords

Apoptosis; B lymphocyte; Caspase; Free radicals; Hydrogen peroxide

Indexed keywords

ANTIOXIDANT; B LYMPHOCYTE RECEPTOR; BUTHIONINE SULFOXIMINE; CATALASE; FAS ANTIGEN; HYDROGEN PEROXIDE; INHIBITOR OF APOPTOSIS PROTEIN; REACTIVE OXYGEN METABOLITE; SUPEROXIDE; SUPEROXIDE DISMUTASE;

ANIMAL CELL; ANTIOXIDANT ACTIVITY; APOPTOSIS; ARTICLE; B CELL LYMPHOMA; B LYMPHOCYTE; CELL PROTECTION; CONTROLLED STUDY; CORRELATION ANALYSIS; CROSS LINKING; DEPLETION; ENZYME ACTIVATION; MOUSE; NONHUMAN; OXIDATION REDUCTION REACTION; OXIDATIVE STRESS; PHENOTYPE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; RECEPTOR INTRINSIC ACTIVITY; RECEPTOR SENSITIVITY;

EID: 0042328267     PISSN: 08915849     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0891-5849(03)00391-5     Document Type: Article

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