Apoptosis signaling in lymphocytes

Current Opinion in Immunology

Volumn 11, Issue 3, 1999, Pages 277-285

  Peter, Marcus E ^a  

^a NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CASPASE; COMPLEMENTARY DNA; CYTOCHROME C; INTERLEUKIN 2; INTERLEUKIN 2 RECEPTOR; LYMPHOCYTE ANTIGEN RECEPTOR; LYMPHOCYTE RECEPTOR; MEMBRANE COFACTOR PROTEIN; PROTEIN BAX; PROTEIN BCL 2; TRANSCRIPTION FACTOR; TUMOR NECROSIS FACTOR RECEPTOR;

APOPTOSIS; AUTOIMMUNITY; B LYMPHOCYTE; GENE ISOLATION; HOMEOSTASIS; IMMUNE RESPONSE; IMMUNOCOMPETENT CELL; MOLECULAR BIOLOGY; NONHUMAN; REVIEW; SIGNAL TRANSDUCTION; T LYMPHOCYTE; THYMOCYTE;

EID: 0033036748     PISSN: 09527915     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0952-7915(99)80045-4     Document Type: Article

References (3)

1. Metzstein MM, Stanfield GM, Horvitz HR: Genetics of programmed cell death in C. elegans: past, present and future. Trends Genet 1998, 14:410-416.
2. Zou H, Henzel WJ, Liu X, Lutschg A, Wang X: Apaf-1, a human protein homologous to C. elegans CED-4, participates in cytochrome c-dependent activation of caspase-3. Cell 1997, 90:405-413.
3. Conradt B, Horvitz HR: The C. elegans protein EGL-1 is required for programmed cell death and interacts with the Bcl-2-like protein CED-9. Cell 1998, 93:519-529. In this report egl-1, which encodes a BHS-containing CED-9 homolog, is identified as a fourth gene of C. elegans required for execution of apoptosis during development of the worm.