H2O2 regulates cardiac myocyte phenotype via concentration-dependent activation of distinct kinase pathways

Journal of Molecular and Cellular Cardiology

Volumn 35, Issue 6, 2003, Pages 615-621

  Kwon, Susan H ^a   Pimentel, David R ^a   Remondino, Andrea ^a   Sawyer, Douglas B ^a   Colucci, Wilson S ^a  

^a BOSTON UNIVERSITY MEDICAL CENTER   (United States)

Author keywords

Akt; Apoptosis; c Jun; Cardiac myocyte; ERK1 2; H2O2; Hypertrophy; JNK; p38 kinase; Reactive oxygen species

Indexed keywords

1,4 DIAMINO 1,4 BIS(2 AMINOPHENYLTHIO) 2,3 DICYANOBUTADIENE; HYDROGEN PEROXIDE; MITOGEN ACTIVATED PROTEIN KINASE 1; PROTEIN KINASE; PROTEIN KINASE B; PROTEIN KINASE P38; UNCLASSIFIED DRUG;

ADENOVIRUS; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL DEATH; CELL SURVIVAL; CONCENTRATION (PARAMETERS); CONCENTRATION RESPONSE; CONTROLLED STUDY; EXPERIMENTAL RAT; EXPOSURE; HEART HYPERTROPHY; HEART MUSCLE CELL; MALE; NICK END LABELING; NONHUMAN; PHENOTYPE; PRIORITY JOURNAL; PROTEIN SYNTHESIS; RAT; SIGNAL TRANSDUCTION;

ADENOVIRIDAE;

EID: 0037905740     PISSN: 00222828     EISSN: None     Source Type: Journal    
DOI: 10.1016/S0022-2828(03)00084-1     Document Type: Article

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