-
1
-
-
0027490362
-
Giant multilevel cation channels formed by Alzheimer disease amyloid beta-protein [A beta P-(1-40)] in bilayer membranes
-
ARISPE N., POLLARD H. B., ROJAS E. Giant multilevel cation channels formed by Alzheimer disease amyloid beta-protein [A beta P-(1-40)] in bilayer membranes. Proc. Natl. Acad. Sci. U.S.A. 90:1993a;10 573-10 577.
-
(1993)
Proc. Natl. Acad. Sci. U.S.A.
, vol.90
, pp. 10573-10577
-
-
ARISPE, N.1
POLLARD H., B.2
ROJAS, E.3
-
2
-
-
0028649480
-
The ability of amyloid beta-protein [A Beta P (1-40)] to form Ca2+ channels provides a mechanism for neuronal death in Alzheimer's disease
-
ARISPE N., POLLARD H. B., ROJAS E. The ability of amyloid beta-protein [A Beta P (1-40)] to form Ca2+ channels provides a mechanism for neuronal death in Alzheimer's disease. Ann. N. Y. Acad. Sci. 747:1994a;256-266.
-
(1994)
Ann. N. Y. Acad. Sci.
, vol.747
, pp. 256-266
-
-
ARISPE, N.1
POLLARD H., B.2
ROJAS, E.3
-
3
-
-
0028670272
-
Beta-Amyloid Ca(2+)-channel hypothesis for neuronal death in Alzheimer disease
-
ARISPE N., POLLARD H. B., ROJAS E. Beta-Amyloid Ca(2+)-channel hypothesis for neuronal death in Alzheimer disease. Mol. Cell. Biochem. 140:1994b;119-125.
-
(1994)
Mol. Cell. Biochem.
, vol.140
, pp. 119-125
-
-
ARISPE, N.1
POLLARD H., B.2
ROJAS, E.3
-
4
-
-
0027508926
-
Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: Blockade by tromethamine and aluminum
-
ARISPE N., ROJAS E., POLLARD H. B. Alzheimer disease amyloid beta protein forms calcium channels in bilayer membranes: blockade by tromethamine and aluminum. Proc. Natl. Acad. Sci. U.S.A. 90:1993b;567-571.
-
(1993)
Proc. Natl. Acad. Sci. U.S.A.
, vol.90
, pp. 567-571
-
-
ARISPE, N.1
ROJAS, E.2
POLLARD H., B.3
-
5
-
-
0030949517
-
Amyloid beta-peptides increase annular and bulk fluidity and induce lipid peroxidation in brain synaptic plasma membranes
-
AVDULOV N. A., CHOCHINA S. V., IGBAVBOA U., O'HARE E. O., SCHROEDER F., CLEARY J. P., WOOD W. G. Amyloid beta-peptides increase annular and bulk fluidity and induce lipid peroxidation in brain synaptic plasma membranes. J Neurochem. 68:1997;2086-2091.
-
(1997)
J Neurochem.
, vol.68
, pp. 2086-2091
-
-
AVDULOV N., A.1
CHOCHINA S., V.2
IGBAVBOA, U.3
O'HARE E., O.4
SCHROEDER, F.5
CLEARY J., P.6
WOOD W., G.7
-
6
-
-
0033103343
-
The amyloid precursor protein interacts with Go heterotrimeric protein within a cell compartment specialized in signal transduction
-
BROUILLET E., TREMBLEAU A., GALANAUD D., VOLOVITCH M., BOUILLOT C., VALENZA C., PROCHIANTZ A., ALLINQUANT B. The amyloid precursor protein interacts with Go heterotrimeric protein within a cell compartment specialized in signal transduction. J. Neurosci. 19:1999;1717-1727.
-
(1999)
J. Neurosci.
, vol.19
, pp. 1717-1727
-
-
BROUILLET, E.1
TREMBLEAU, A.2
GALANAUD, D.3
VOLOVITCH, M.4
BOUILLOT, C.5
VALENZA, C.6
PROCHIANTZ, A.7
ALLINQUANT, B.8
-
7
-
-
0028588694
-
Congo red protects against toxicity of beta-amyloid peptides on rat hippocampal neurones
-
BURGEVIN M. C., PASSAT M., DANIEL N., CAPET M., DOBLE A. Congo red protects against toxicity of beta-amyloid peptides on rat hippocampal neurones. Neuroreport. 5:1994;2429-2432.
-
(1994)
Neuroreport
, vol.5
, pp. 2429-2432
-
-
BURGEVIN M., C.1
PASSAT, M.2
DANIEL, N.3
CAPET, M.4
DOBLE, A.5
-
8
-
-
0027429732
-
Beta-amyloid neurotoxicity in human cortical culture is not mediated by excitotoxins
-
BUSCIGLIO J., YEH J., YANKNER B. A. Beta-amyloid neurotoxicity in human cortical culture is not mediated by excitotoxins. J. Neurochem. 61:1993;1565-1568.
-
(1993)
J. Neurochem.
, vol.61
, pp. 1565-1568
-
-
BUSCIGLIO, J.1
YEH, J.2
YANKNER B., A.3
-
9
-
-
0030928406
-
Beta-amyloid-associated free radical oxidative stress and neurotoxicity: Implica-tions for Alzheimer's disease
-
BUTTERFIELD D. A. Beta-amyloid-associated free radical oxidative stress and neurotoxicity: implica-tions for Alzheimer's disease. Chem. Res. Toxicol. 10:1997;495-506.
-
(1997)
Chem. Res. Toxicol.
, vol.10
, pp. 495-506
-
-
BUTTERFIELD D., A.1
-
10
-
-
0028178837
-
Beta-amyloid peptide free radical fragments initiate synaptosomal lipoperoxidation in a sequence-specific fashion: Implications to Alzheimer's disease
-
BUTTERFIELD D. A., HENSLEY K., HARRIS M., MATTSON M., CARNEY J. Beta-amyloid peptide free radical fragments initiate synaptosomal lipoperoxidation in a sequence-specific fashion: implications to Alzheimer's disease. Biochem. Biophys. Res. Commun. 200:1994;710-715.
-
(1994)
Biochem. Biophys. Res. Commun.
, vol.200
, pp. 710-715
-
-
BUTTERFIELD D., A.1
HENSLEY, K.2
HARRIS, M.3
MATTSON, M.4
CARNEY, J.5
-
11
-
-
0032867538
-
Amyloid beta-peptide-associated free radical oxidative stress, neurotoxicity, and Alzheimer's disease
-
BUTTERFIELD D. A., YATIN S. M., VARADARAJAN S., KOPPAL T. Amyloid beta-peptide-associated free radical oxidative stress, neurotoxicity, and Alzheimer's disease. Methods Enzymol. 309:1999;746-768.
-
(1999)
Methods Enzymol.
, vol.309
, pp. 746-768
-
-
BUTTERFIELD D., A.1
YATIN S., M.2
VARADARAJAN, S.3
KOPPAL, T.4
-
12
-
-
0034125199
-
Cholesterol modulates the membrane-disordering effects of beta-amyloid peptides in the hippocampus: Specific changes in Alzheimer's disease
-
ECKERT G. P., CAIRNS N. J., MARAS A., GATTAZ W. F., MULLER W. E. Cholesterol modulates the membrane-disordering effects of beta-amyloid peptides in the hippocampus: specific changes in Alzheimer's disease. Dement. Geriatr. Cogn. Disord. 11:2000;181-186.
-
(2000)
Dement. Geriatr. Cogn. Disord.
, vol.11
, pp. 181-186
-
-
ECKERT G., P.1
CAIRNS N., J.2
MARAS, A.3
GATTAZ W., F.4
MULLER W., E.5
-
13
-
-
0028662013
-
Molecular mechanisms of memory and the pathophysiology of Alzheimer's disease
-
ETCHEBERRIGARAY E., GIBSON G. E., ALKON D. L. Molecular mechanisms of memory and the pathophysiology of Alzheimer's disease. Ann. N. Y. Acad. Sci. 747:1994;245-255.
-
(1994)
Ann. N. Y. Acad. Sci.
, vol.747
, pp. 245-255
-
-
ETCHEBERRIGARAY, E.1
GIBSON G., E.2
ALKON D., L.3
-
15
-
-
0030451766
-
Effect of beta-amyloid block of the fast-inactivating K+ channel on intracellular Ca2+ and excitability in a modeled neuron
-
GOOD T. A., MURPHY R. M. Effect of beta-amyloid block of the fast-inactivating K+ channel on intracellular Ca2+ and excitability in a modeled neuron. Proc. Natl. Acad. Sci. U.S.A. 93:1996;15130-15135.
-
(1996)
Proc. Natl. Acad. Sci. U.S.A.
, vol.93
, pp. 15130-15135
-
-
GOOD T., A.1
MURPHY R., M.2
-
16
-
-
0029670616
-
Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons
-
GOOD T. A., SMITH D. O., MURPHY R. M. Beta-amyloid peptide blocks the fast-inactivating K+ current in rat hippocampal neurons. Biophys. J. 70:1996;296-304.
-
(1996)
Biophys. J.
, vol.70
, pp. 296-304
-
-
GOOD T., A.1
SMITH D., O.2
MURPHY R., M.3
-
17
-
-
0033740610
-
Mechanisms of beta-amyloid neurotoxicity: Perspectives of pharmacotherapy
-
HARKANY T., ABRAHAM I., KONYA C., NYAKAS C., ZARANDI M., PENKE B., LUITEN P. G. Mechanisms of beta-amyloid neurotoxicity: perspectives of pharmacotherapy. Rev. Neurosci. 11:2000;329-382.
-
(2000)
Rev. Neurosci.
, vol.11
, pp. 329-382
-
-
HARKANY, T.1
ABRAHAM, I.2
KONYA, C.3
NYAKAS, C.4
ZARANDI, M.5
PENKE, B.6
LUITEN P., G.7
-
18
-
-
0033570337
-
Protofibrillar intermediates of amyloid beta-protein induce acute electrophysiological changes and progressive neurotoxicity in cortical neurons
-
HARTLEY D. M., WALSH D. M., YE C. P., DIEHL T., VASQUEZ S., VASSILEV P. M., TEPLOW D. B., SELKOE D. J. Protofibrillar intermediates of amyloid beta-protein induce acute electrophysiological changes and progressive neurotoxicity in cortical neurons. J. Neurosci. 19:1999;8876-8884.
-
(1999)
J. Neurosci.
, vol.19
, pp. 8876-8884
-
-
HARTLEY D., M.1
WALSH D., M.2
YE C., P.3
DIEHL, T.4
VASQUEZ, S.5
VASSILEV P., M.6
TEPLOW D., B.7
SELKOE D., J.8
-
19
-
-
0029034927
-
Aggregation state and neurotoxic properties of Alzheimer beta-amyloid peptide
-
HOWLETT D. R., JENNINGS K. H., LEE D. C., CLARK M. S., BROWN F., WETZEL R., WOOD S. J., CAMILLERI P., ROBERTS G. W. Aggregation state and neurotoxic properties of Alzheimer beta-amyloid peptide. Neurodegeneration. 4:1995;23-32.
-
(1995)
Neurodegeneration
, vol.4
, pp. 23-32
-
-
HOWLETT D., R.1
JENNINGS K., H.2
LEE D., C.3
CLARK M., S.4
BROWN, F.5
WETZEL, R.6
WOOD S., J.7
CAMILLERI, P.8
ROBERTS G., W.9
-
20
-
-
0035054836
-
Copernicus revisited: Amyloid beta in Alzheimer's disease
-
JOSEPH J., SHUKITT-HALE B., DENISOVA N. A., MARTIN A., PERRY G., SMITH M. A. Copernicus revisited: amyloid beta in Alzheimer's disease. Neurobiol Aging. 22:2001;131-146.
-
(2001)
Neurobiol Aging
, vol.22
, pp. 131-146
-
-
JOSEPH, J.1
SHUKITT-HALE, B.2
DENISOVA N., A.3
MARTIN, A.4
PERRY, G.5
SMITH M., A.6
-
21
-
-
0032699297
-
Amyloid beta peptide membrane perturbation is the basis for its biological effects
-
KANFER J. N., SORRENTINO G., SITAR D. S. Amyloid beta peptide membrane perturbation is the basis for its biological effects. Neurochem. Res. 24:1999;1621-1630.
-
(1999)
Neurochem. Res.
, vol.24
, pp. 1621-1630
-
-
KANFER J., N.1
SORRENTINO, G.2
SITAR D., S.3
-
22
-
-
0023105114
-
The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor
-
KANG J., LEMAIRE H. G., UNTERBECK A., SALBAUM J. M., MASTERS C. L., GRZESCHIK K. H., MULTHAUP G., BEYREUTHER K., MULLER-HILL B. The precursor of Alzheimer's disease amyloid A4 protein resembles a cell-surface receptor. Nature. 325:1987;733-736.
-
(1987)
Nature
, vol.325
, pp. 733-736
-
-
KANG, J.1
LEMAIRE H., G.2
UNTERBECK, A.3
SALBAUM J., M.4
MASTERS C., L.5
GRZESCHIK K., H.6
MULTHAUP, G.7
BEYREUTHER, K.8
MULLER-HILL, B.9
-
23
-
-
0030602845
-
Activation of pertussis toxin-sensitive G-proteins in membranes of SH-SY5Y human neuroblastoma cells and bovine transducin by ethanol
-
KLINKER J. F., LICHTENBERG-KRAAG B., DAMM H., ROMMELSPACHER H. Activation of pertussis toxin-sensitive G-proteins in membranes of SH-SY5Y human neuroblastoma cells and bovine transducin by ethanol. Neurosci. Lett. 213:1996;25-28.
-
(1996)
Neurosci. Lett.
, vol.213
, pp. 25-28
-
-
KLINKER J., F.1
LICHTENBERG-KRAAG, B.2
DAMM, H.3
ROMMELSPACHER, H.4
-
24
-
-
0025110182
-
Beta-amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage
-
KOH J. Y., YANG L. L., COTMAN C. W. Beta-amyloid protein increases the vulnerability of cultured cortical neurons to excitotoxic damage. Brain Res. 533:1990;315-320.
-
(1990)
Brain Res.
, vol.533
, pp. 315-320
-
-
KOH J., Y.1
YANG L., L.2
COTMAN C., W.3
-
25
-
-
0034702813
-
Correlation of beta-amyloid aggregate size and hydrophobicity with decreased bilayer fluidity of model membranes
-
KREMER J. J., PALLITTO M. M., SKLANSKY D. J., MURPHY R. M. Correlation of beta-amyloid aggregate size and hydrophobicity with decreased bilayer fluidity of model membranes. Biochemistry. 39:2000;10309-10318.
-
(2000)
Biochemistry
, vol.39
, pp. 10309-10318
-
-
KREMER J., J.1
PALLITTO M., M.2
SKLANSKY D., J.3
MURPHY R., M.4
-
26
-
-
0035942997
-
Profile of changes in lipid bilayer structure caused by beta-amyloid peptide
-
KREMER J. J., SKLANSKY D. J., MURPHY R. M. Profile of changes in lipid bilayer structure caused by beta-amyloid peptide. Biochemistry. 40:2001;8563-8571.
-
(2001)
Biochemistry
, vol.40
, pp. 8563-8571
-
-
KREMER J., J.1
SKLANSKY D., J.2
MURPHY R., M.3
-
27
-
-
11544279355
-
Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins
-
LAMBERT M. P., BARLOW A. K., CHROMY B. A., EDWARDS C., FREED R., LIOSATOS M., MORGAN T. E., ROZOVSKY I., TROMMER B., VIOLA K. L., WALS P., ZHANG C., FINCH C. E., KRAFFT G. A., KLEIN W. L. Diffusible, nonfibrillar ligands derived from Abeta1-42 are potent central nervous system neurotoxins. Proc. Natl. Acad. Sci. U.S.A. 95:1998;6448-6453.
-
(1998)
Proc. Natl. Acad. Sci. U.S.A.
, vol.95
, pp. 6448-6453
-
-
LAMBERT M., P.1
BARLOW A., K.2
CHROMY B., A.3
EDWARDS, C.4
FREED, R.5
LIOSATOS, M.6
MORGAN T., E.7
ROZOVSKY, I.8
TROMMER, B.9
VIOLA K., L.10
WALS, P.11
ZHANG, C.12
FINCH C., E.13
KRAFFT G., A.14
KLEIN W., L.15
-
28
-
-
0028172886
-
Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red
-
LORENZO A., YANKNER B. A. Beta-amyloid neurotoxicity requires fibril formation and is inhibited by congo red. Proc. Natl. Acad. Sci. U.S.A. 91:1994;12243-12247.
-
(1994)
Proc. Natl. Acad. Sci. U.S.A.
, vol.91
, pp. 12243-12247
-
-
LORENZO, A.1
YANKNER B., A.2
-
30
-
-
0032014429
-
Effects of beta-amyloid peptides on the fluidity of membranes from frontal and parietal lobes of human brain. High potencies of A beta 1-42 and A beta 1-43
-
MULLER W. E., ECKERT G. P., SCHEUER K., CAIRNS N. J., MARAS A., GATTAZ W. F. Effects of beta-amyloid peptides on the fluidity of membranes from frontal and parietal lobes of human brain. High potencies of A beta 1-42 and A beta 1-43. Amyloid. 5:1998;10-15.
-
(1998)
Amyloid
, vol.5
, pp. 10-15
-
-
MULLER W., E.1
ECKERT G., P.2
SCHEUER, K.3
CAIRNS N., J.4
MARAS, A.5
GATTAZ W., F.6
-
31
-
-
0028987055
-
Beta-amyloid peptide decreases membrane fluidity
-
MULLER W. E., KOCH S., ECKERT A., HARTMANN H., SCHEUER K. Beta-amyloid peptide decreases membrane fluidity. Brain Res. 674:1995;133-136.
-
(1995)
Brain Res.
, vol.674
, pp. 133-136
-
-
MULLER W., E.1
KOCH, S.2
ECKERT, A.3
HARTMANN, H.4
SCHEUER, K.5
-
32
-
-
0033596944
-
Recognition sequence design for peptidyl modulators of beta-amyloid aggregation and toxicity
-
PALLITTO M. M., GHANTA J., HEINZELMAN P., KIESSLING L. L., MURPHY R. M. Recognition sequence design for peptidyl modulators of beta-amyloid aggregation and toxicity. Biochemistry. 38:1999;3570-3578.
-
(1999)
Biochemistry
, vol.38
, pp. 3570-3578
-
-
PALLITTO M., M.1
GHANTA, J.2
HEINZELMAN, P.3
KIESSLING L., L.4
MURPHY R., M.5
-
33
-
-
0028870182
-
Sulfated glycosaminoglycans and dyes attenuate the neurotoxic effects of beta-amyloid in rat PC12 cells
-
POLLACK S. J., SADLER I. I., HAWTIN S. R., TAILOR V. J., SHEARMAN M. S. Sulfated glycosaminoglycans and dyes attenuate the neurotoxic effects of beta-amyloid in rat PC12 cells. Neurosci. Lett. 184:1995;113-116.
-
(1995)
Neurosci. Lett.
, vol.184
, pp. 113-116
-
-
POLLACK S., J.1
SADLER I., I.2
HAWTIN S., R.3
TAILOR V., J.4
SHEARMAN M., S.5
-
34
-
-
0035951774
-
The role of G protein activation in the toxicity of amyloidogenic Abeta- (1-40), Abeta-(25-35), and bovine calcitonin
-
RYMER D. L., GOOD T. A. The role of G protein activation in the toxicity of amyloidogenic Abeta- (1-40), Abeta-(25-35), and bovine calcitonin. J. Biol. Chem. 276:2001;2523-2530.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 2523-2530
-
-
RYMER D., L.1
GOOD T., A.2
-
35
-
-
0029549796
-
Sulphated compounds attenuate beta-amyloid toxicity by inhibiting its association with cells
-
SADLER I. I., SMITH D. W., SHEARMAN M. S., RAGAN C. I., TAILOR V. J., POLLACK S. J. Sulphated compounds attenuate beta-amyloid toxicity by inhibiting its association with cells. Neuroreport. 7:1995;49-53.
-
(1995)
Neuroreport
, vol.7
, pp. 49-53
-
-
SADLER I., I.1
SMITH D., W.2
SHEARMAN M., S.3
RAGAN C., I.4
TAILOR V., J.5
POLLACK S., J.6
-
36
-
-
0028224211
-
Adenylyl cyclase activity in Alzheimer's disease brain: Stimulatory and inhibitory signal transduction pathways are differently affected
-
SCHNECKO A., WITTE K., BOHL J., OHM T., LEMMER B. Adenylyl cyclase activity in Alzheimer's disease brain: stimulatory and inhibitory signal transduction pathways are differently affected. Brain Res. 644:1994;291-296.
-
(1994)
Brain Res.
, vol.644
, pp. 291-296
-
-
SCHNECKO, A.1
WITTE, K.2
BOHL, J.3
OHM, T.4
LEMMER, B.5
-
37
-
-
0031170886
-
The toxicity of the Alzheimer's beta-amyloid peptide correlates with a distinct fiber morphology
-
SEILHEIMER B., BOHRMANN B., BONDOLFI L., MULLER F., STUBER D., DOBELI H. The toxicity of the Alzheimer's beta-amyloid peptide correlates with a distinct fiber morphology. J. Struct. Biol. 119:1997;59-71.
-
(1997)
J. Struct. Biol.
, vol.119
, pp. 59-71
-
-
SEILHEIMER, B.1
BOHRMANN, B.2
BONDOLFI, L.3
MULLER, F.4
STUBER, D.5
DOBELI, H.6
-
38
-
-
0028123071
-
Alzheimer's disease: A central role for amyloid [see comments]
-
SELKOE D. J. Alzheimer's disease: a central role for amyloid [see comments]. J. Neuropathol. Exp. Neurol. 53:1994;438-447.
-
(1994)
J. Neuropathol. Exp. Neurol.
, vol.53
, pp. 438-447
-
-
SELKOE D., J.1
-
39
-
-
17544404378
-
(-)Nicotine inhibits the activations of phospholipases A2 and D by amyloid beta peptide
-
SINGH I. N., SORRENTINO G., SITAR D. S., KANFER J. N. (-)Nicotine inhibits the activations of phospholipases A2 and D by amyloid beta peptide. Brain Res. 800:1998;275-281.
-
(1998)
Brain Res.
, vol.800
, pp. 275-281
-
-
SINGH I., N.1
SORRENTINO, G.2
SITAR D., S.3
KANFER J., N.4
-
40
-
-
0032758498
-
Regulation of GTPase and adenylate cyclase activity by amyloid beta- peptide and its fragments in rat brain tissue
-
SOOMETS U., MAHLAPUU R., TEHRANIAN R., JARVET J., KARELSON E., ZILMER M., IVERFELDT K., ZORKO M., GRASLUND A., LANGEL U. Regulation of GTPase and adenylate cyclase activity by amyloid beta- peptide and its fragments in rat brain tissue. Brain Res. 850:1999;179-188.
-
(1999)
Brain Res.
, vol.850
, pp. 179-188
-
-
SOOMETS, U.1
MAHLAPUU, R.2
TEHRANIAN, R.3
JARVET, J.4
KARELSON, E.5
ZILMER, M.6
IVERFELDT, K.7
ZORKO, M.8
GRASLUND, A.9
LANGEL, U.10
-
41
-
-
0033860372
-
Review: Alzheimer's amyloid beta-peptide-associated free radical oxidative stress and neurotoxicity [In Process Citation]
-
VARADARAJAN S., YATIN S., AKSENOVA M., BUTTERFIELD D. A. Review: Alzheimer's amyloid beta-peptide-associated free radical oxidative stress and neurotoxicity [In Process Citation]. J. Struct. Biol. 130:2000;184-208.
-
(2000)
J. Struct. Biol.
, vol.130
, pp. 184-208
-
-
VARADARAJAN, S.1
YATIN, S.2
AKSENOVA, M.3
BUTTERFIELD D., A.4
-
42
-
-
0033520461
-
Amyloid beta-protein fibrillogenesis. Structure and biological activity of protofibrillar intermediates
-
WALSH D. M., HARTLEY D. M., KUSUMOTO Y., FEZOUI Y., CONDRON M. M., LOMAKIN A., BENEDEK G. B., SELKOE D. J., TEPLOW D. B. Amyloid beta-protein fibrillogenesis. Structure and biological activity of protofibrillar intermediates. J. Biol. Chem. 274:1999;25945-25952.
-
(1999)
J. Biol. Chem.
, vol.274
, pp. 25945-25952
-
-
WALSH D., M.1
HARTLEY D., M.2
KUSUMOTO, Y.3
FEZOUI, Y.4
CONDRON M., M.5
LOMAKIN, A.6
BENEDEK G., B.7
SELKOE D., J.8
TEPLOW D., B.9
-
43
-
-
0035834692
-
Reduction in cholesterol and sialic acid content protects cells from the toxic effects of beta -amyloid peptides
-
WANG S. S., RYMER D. L., GOOD T. A. Reduction in cholesterol and sialic acid content protects cells from the toxic effects of beta -amyloid peptides. J. Biol. Chem. 276:2001;42027-42034.
-
(2001)
J. Biol. Chem.
, vol.276
, pp. 42027-42034
-
-
WANG S., S.1
RYMER D., L.2
GOOD T., A.3
-
44
-
-
0037026929
-
Development of a novel diffusion-based method to estimate the size of the aggregated Ab species responsible for neurotoxicity
-
WANG S. S., BECERRA-ARTEAGA A., GOOD T. A. Development of a novel diffusion-based method to estimate the size of the aggregated Ab species responsible for neurotoxicity. Biotechnol Bioeng. 80:2002;50-59.
-
(2002)
Biotechnol Bioeng.
, vol.80
, pp. 50-59
-
-
WANG S., S.1
BECERRA-ARTEAGA, A.2
GOOD T., A.3
-
45
-
-
0034658615
-
Fractionation and characterization of oligomeric, protofibrillar and fibrillar forms of beta-amyloid peptide
-
WARD R. V., JENNINGS K. H., JEPRAS R., NEVILLE W., OWEN D. E., HAWKINS J., CHRISTIE G., DAVIS J. B., GEORGE A., KARRAN E. H., HOWLETT D. R. Fractionation and characterization of oligomeric, protofibrillar and fibrillar forms of beta-amyloid peptide. Biochem. J. 348 Pt 1:2000;137-144.
-
(2000)
Biochem. J.
, vol.348
, Issue.PART 1
, pp. 137-144
-
-
WARD R., V.1
JENNINGS K., H.2
JEPRAS, R.3
NEVILLE, W.4
OWEN D., E.5
HAWKINS, J.6
CHRISTIE, G.7
DAVIS J., B.8
GEORGE, A.9
KARRAN E., H.10
HOWLETT D., R.11
-
46
-
-
0028180304
-
Ca2+ channel blockers attenuate beta-amyloid peptide toxicity to cortical neurons in culture
-
WEISS J. H., PIKE C. J., COTMAN C. W. Ca2+ channel blockers attenuate beta-amyloid peptide toxicity to cortical neurons in culture. J. Neurochem. 62:1994;372-375.
-
(1994)
J. Neurochem.
, vol.62
, pp. 372-375
-
-
WEISS J., H.1
PIKE C., J.2
COTMAN C., W.3
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