IL-18 contributes to the spontaneous development of atopic dermatitis-like inflammatory skin lesion independently of IgE/stat6 under specific pathogen-free conditions

Proceedings of the National Academy of Sciences of the United States of America

Volumn 99, Issue 17, 2002, Pages 11340-11345

  Konishi, Hiroshi ^a   Tsutsui, Hiroko ^a   Murakami, Takaaki ^a   Yumikura Futatsugi, Shizue ^a   Yamanaka, Kei ichi ^a   Tanaka, Minoru ^a   Iwakura, Yohichiro ^a   Suzuki, Noboru ^a   Takeda, Kiyoshi ^a   Akira, Shizuo ^a   Nakanishi, Kenji ^a   Mizutani, Hitoshi ^a  

^a HYOGO COLLEGE OF MEDICINE   (Japan)

Author keywords

[No Author keywords available]

Indexed keywords

HISTAMINE; IMMUNOGLOBULIN E; INTERLEUKIN 18; INTERLEUKIN 1BETA CONVERTING ENZYME; STAT6 PROTEIN;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; ATOPIC DERMATITIS; CONTROLLED STUDY; CYTOKINE PRODUCTION; CYTOKINE RELEASE; FEMALE; HISTAMINE RELEASE; MALE; MAST CELL; MASTOCYTOSIS; MOUSE; NEUTROPHIL; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SKIN INFLAMMATION; T LYMPHOCYTE; TH2 CELL;

ANIMALS; DERMATITIS, ATOPIC; HUMANS; IMMUNOGLOBULIN E; INFLAMMATION; INTERLEUKIN-18; KERATINS; MICE; MICE, TRANSGENIC; PROMOTER REGIONS (GENETICS); PRURITUS; RABBITS; SIGNAL TRANSDUCTION; SPECIFIC PATHOGEN-FREE ORGANISMS; STAT6 TRANSCRIPTION FACTOR; T-LYMPHOCYTES; TH2 CELLS; TRANS-ACTIVATORS;

ANIMALIA;

EID: 0037143717     PISSN: 00278424     EISSN: None     Source Type: Journal    
DOI: 10.1073/pnas.152337799     Document Type: Article

References (44)