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Volumn 21, Issue 6, 2002, Pages 1447-1455
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Targeted disruption of NBS1 reveals its roles in mouse development and DNA repair
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Author keywords
Cancer; Cell cycle; DNA repair; Genetic stability; Immunodeficiency
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Indexed keywords
GENE PRODUCT;
NIBRIN;
UNCLASSIFIED DRUG;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ANIMAL TISSUE;
ARTICLE;
CELL MATURATION;
CELL PROLIFERATION;
CONTROLLED STUDY;
DNA REPAIR;
DNA STRAND BREAKAGE;
FEMALE;
FEMALE INFERTILITY;
GENE ACTIVITY;
GENE DISRUPTION;
GENE MUTATION;
GERM CELL;
GROWTH RETARDATION;
MOUSE;
NIJMEGEN BREAKAGE SYNDROME;
NONHUMAN;
PRIORITY JOURNAL;
RADIOSENSITIVITY;
SPERMATOGENESIS;
THYMUS LYMPHOMA;
ANIMALS;
ATAXIA TELANGIECTASIA;
CELL CYCLE PROTEINS;
CELL DIVISION;
CELLS, CULTURED;
DNA;
DNA REPAIR;
DNA-BINDING PROTEINS;
FEMALE;
G2 PHASE;
GAMMA RAYS;
GENE TARGETING;
GROWTH;
IMMUNOLOGIC DEFICIENCY SYNDROMES;
LYMPHOID TISSUE;
LYMPHOMA;
MALE;
MICE;
MICE, KNOCKOUT;
NUCLEAR PROTEINS;
OOCYTES;
OOGENESIS;
PROTEIN-SERINE-THREONINE KINASES;
RADIATION TOLERANCE;
SPERMATOGENESIS;
SPERMATOZOA;
THYMUS NEOPLASMS;
TUMOR SUPPRESSOR PROTEINS;
ANIMALIA;
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EID: 0037086702
PISSN: 02614189
EISSN: None
Source Type: Journal
DOI: 10.1093/emboj/21.6.1447 Document Type: Article |
Times cited : (144)
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References (43)
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