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Proceedings of the National Academy of Sciences of the United States of America

Volumn 99, Issue 12, 2002, Pages 8265-8270

Targeted inactivation of CTNNB1 reveals unexpected effects of β-catenin mutation

(5) Chan, Timothy A a Wang, Zhenghe a Dang, Long H a Vogelstein, Bert b Kinzler, Kenneth W a

a JOHNS HOPKINS UNIVERSITY (United States)

b DEPARTMENT OF PATHOLOGY (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BETA CATENIN;

ALLELE; ARTICLE; BIOCHEMISTRY; CANCER CELL CULTURE; CELL FRACTIONATION; CELL GROWTH; CELLULAR DISTRIBUTION; COLON POLYPOSIS; COLONY FORMATION; COLORECTAL CANCER; CONTROLLED STUDY; CTNNB1 GENE; GENE DISRUPTION; GENE ISOLATION; GENE MUTATION; HUMAN; HUMAN CELL; IMMUNOHISTOCHEMISTRY; PRIORITY JOURNAL; PROTEIN LOCALIZATION; PROTEIN TARGETING; WILD TYPE;

BETA CATENIN; CHROMOSOMES, ARTIFICIAL, BACTERIAL; COLORECTAL NEOPLASMS; CYTOSKELETAL PROTEINS; GENE DELETION; GENE EXPRESSION REGULATION, NEOPLASTIC; GENES, APC; HETEROZYGOTE; HUMANS; INTRONS; MUTAGENESIS; RECOMBINANT PROTEINS; RNA PROCESSING, POST-TRANSCRIPTIONAL; TRANS-ACTIVATORS; TRANSCRIPTION, GENETIC; TRANSFECTION; TUMOR CELLS, CULTURED; TUMOR STEM CELL ASSAY;

EID: 0037062430 PISSN: 00278424 EISSN: None Source Type: Journal
DOI: 10.1073/pnas.082240999 Document Type: Article

Times cited : (118)

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