|
Volumn 22, Issue 19, 2002, Pages 6767-6778
|
Inactivation of the retinoblastoma protein family can bypass the HCF-1 defect in tsBN67 cell proliferation and cytokinesis
a a a |
Author keywords
[No Author keywords available]
|
Indexed keywords
ONCOPROTEIN;
RETINOBLASTOMA PROTEIN;
VIRUS T ANTIGEN;
ADENOVIRUS;
ANIMAL CELL;
ARTICLE;
CELL DAMAGE;
CELL PROLIFERATION;
CELL PROLIFERATION FACTOR 1;
CELL STRAIN;
CYTOKINESIS;
MAMMAL CELL;
NONHUMAN;
PRIORITY JOURNAL;
PROTEIN FUNCTION;
SIMIAN VIRUS 40;
TEMPERATURE;
ADENOVIRUS E1A PROTEINS;
AMINO ACID MOTIFS;
ANIMALS;
ANTIGENS, VIRAL, TUMOR;
CELL CYCLE PROTEINS;
CELL DIVISION;
CELL LINE;
CHROMATIN;
CRICETINAE;
DNA-BINDING PROTEINS;
E2F TRANSCRIPTION FACTORS;
HOST CELL FACTOR C1;
KIDNEY;
MUTATION, MISSENSE;
NUCLEAR PROTEINS;
ONCOGENE PROTEINS;
PHOSPHOPROTEINS;
PROTEIN BINDING;
PROTEINS;
RETINOBLASTOMA PROTEIN;
RETINOBLASTOMA-LIKE PROTEIN P130;
S PHASE;
SIMIAN VIRUS 40;
TEMPERATURE;
TRANSCRIPTION FACTORS;
TRANSFECTION;
ADENOVIRIDAE;
ANIMALIA;
CRICETINAE;
MAMMALIA;
SIMIAE;
SIMIAN VIRUS 40;
|
EID: 0036785541
PISSN: 02707306
EISSN: None
Source Type: Journal
DOI: 10.1128/MCB.22.19.6767-6778.2002 Document Type: Article |
Times cited : (23)
|
References (44)
|