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Volumn 69, Issue 3, 2002, Pages 362-372
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p35/Cdk5 pathway mediates soluble amyloid-β peptide-induced tau phosphorylation in vitro
a a a a a a a a a a a |
Author keywords
Alzheimer's disease; Amyloid ; Calcium; Calpain; N2a cells; Tau
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Indexed keywords
AMYLOID BETA PROTEIN;
ANTISENSE OLIGONUCLEOTIDE;
CALCIUM CHANNEL L TYPE;
CALPAIN;
CYCLIN DEPENDENT KINASE 5;
PROTEIN P25;
PROTEIN P35;
TAU PROTEIN;
AGING;
ALZHEIMER DISEASE;
CELL DIFFERENTIATION;
CONTROLLED STUDY;
DOG;
DYSTROPHY;
ENZYME ACTIVATION;
ENZYME INHIBITION;
ENZYME MECHANISM;
ENZYME SPECIFICITY;
EPITOPE MAPPING;
HISTOPATHOLOGY;
HUMAN;
HUMAN CELL;
NEUROFIBRILLARY TANGLE;
NEUROPATHOLOGY;
PEPTIDE ANALYSIS;
PRIMATE;
PRIORITY JOURNAL;
PROTEIN PHOSPHORYLATION;
PROTEIN STRUCTURE;
REGULATORY MECHANISM;
SENILE PLAQUE;
SHORT SURVEY;
AMYLOID BETA-PROTEIN;
ANIMALS;
BLOTTING, WESTERN;
CALCIUM CHANNELS, L-TYPE;
CALPAIN;
CELL CULTURE TECHNIQUES;
CYCLIN-DEPENDENT KINASE 5;
CYCLIN-DEPENDENT KINASES;
DOSE-RESPONSE RELATIONSHIP, DRUG;
GENETIC VECTORS;
HUMANS;
IMMUNOHISTOCHEMISTRY;
NERVE TISSUE PROTEINS;
PHOSPHORYLATION;
SIGNAL TRANSDUCTION;
TAU PROTEINS;
TRANSFECTION;
UP-REGULATION;
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EID: 0036682362
PISSN: 03604012
EISSN: None
Source Type: Journal
DOI: 10.1002/jnr.10299 Document Type: Short Survey |
Times cited : (92)
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References (40)
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