Selenium and the regulation of cell signaling, growth, and survival: Molecular and mechanistic aspects

Antioxidants and Redox Signaling

Volumn 4, Issue 2, 2002, Pages 339-351

  McKenzie, Roderick C ^a   Arthur, John R ^a   Beckett, Geoffrey J ^a  

^a UNIVERSITY OF EDINBURGH   (United Kingdom)

Author keywords

[No Author keywords available]

Indexed keywords

ANTIOXIDANT; SELENIUM; SELENOPROTEIN;

ANTIOXIDANT ACTIVITY; CELL DEATH; CELL GROWTH; CELL SURVIVAL; COVALENT BOND; DIETARY INTAKE; GEOGRAPHIC DISTRIBUTION; HISTORY; HUMAN; NONHUMAN; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN SYNTHESIS; REGULATORY MECHANISM; REVIEW; SELENIUM DEFICIENCY; SIGNAL TRANSDUCTION; TOXICITY; TRANSCRIPTION REGULATION;

ANIMALIA;

EID: 0036090765     PISSN: 15230864     EISSN: None     Source Type: Journal    
DOI: 10.1089/152308602753666398     Document Type: Review

References (97)

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14. The thioredoxin and glutathione systems are efficient electron donors to human plasma glutathione peroxidase
15. Interleukin-1-induced nuclear factor kappa B activation is inhibited by overexpression of phospholipid hydroperoxide glutathione peroxidase in a human endothelial cell line
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31. Selenocysteine, identified as the penultimate C-terminal residue in human T-cell thioredoxin reductase, corresponds to TGA in the human placental gene
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68. Deletion of the Saccharomyces cerevisiae TRR1 gene encoding thioredoxin reductase inhibits p53-dependent reporter gene expression
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