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Volumn 509, Issue 1, 2001, Pages 47-52
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Overproduced p73α activates a minimal promoter through a mechanism independent of its transcriptional activity
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Author keywords
P53; p73; Splicing variant; Transcription
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Indexed keywords
PROTEIN P73;
DNA BINDING PROTEIN;
DNA BINDING PROTEINS;
LUCIFERASE;
MESSENGER RNA;
NUCLEAR PROTEIN;
RIBONUCLEASE;
RNA;
ALTERNATIVE RNA SPLICING;
AMINO ACID SEQUENCE;
ARTICLE;
CARBOXY TERMINAL SEQUENCE;
CONTROLLED STUDY;
ENZYME ACTIVATION;
GENE DELETION;
GENE OVEREXPRESSION;
HUMAN;
HUMAN CELL;
PRIORITY JOURNAL;
PROMOTER REGION;
PROTEIN BINDING;
PROTEIN DOMAIN;
TRANSCRIPTION REGULATION;
TUMOR SUPPRESSOR GENE;
BINDING SITE;
BIOSYNTHESIS;
CELL CULTURE;
GENETIC TRANSCRIPTION;
GENETIC TRANSFECTION;
GENETICS;
IMMUNOBLOTTING;
METABOLISM;
PHYSIOLOGY;
PLASMID;
PROTEIN TERTIARY STRUCTURE;
REPORTER GENE;
TRANSACTIVATION;
BINDING SITES;
DNA-BINDING PROTEINS;
GENE DELETION;
GENES, P53;
GENES, REPORTER;
HUMAN;
IMMUNOBLOTTING;
LUCIFERASE;
NUCLEAR PROTEINS;
PLASMIDS;
PROMOTER REGIONS (GENETICS);
PROTEIN BINDING;
PROTEIN STRUCTURE, TERTIARY;
RIBONUCLEASES;
RNA;
RNA, MESSENGER;
SUPPORT, NON-U.S. GOV'T;
TRANS-ACTIVATION (GENETICS);
TRANSCRIPTION, GENETIC;
TRANSFECTION;
TUMOR CELLS, CULTURED;
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EID: 0035976310
PISSN: 00145793
EISSN: None
Source Type: Journal
DOI: 10.1016/S0014-5793(01)03141-6 Document Type: Article |
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Times cited : (6)
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References (38)
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