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Volumn 20, Issue 48, 2001, Pages 7115-7119

Functional mammary gland development and oncogene-induced tumor formation are not affected by the absence of the retinoblastoma gene

(3) Robinson, Gertraud W a Wagner, Kay Uwe a,b Hennighausen, Lothar a

a NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES (United States)

b UNIVERSITY OF NEBRASKA MEDICAL CENTER (United States)

Author keywords

Breast cancer; Mammary gland; Oncogenes; Pocket proteins; Tumor suppressor gene

Indexed keywords

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BREAST CARCINOGENESIS; BREAST DEVELOPMENT; BREAST EPITHELIUM; CANCER SUSCEPTIBILITY; CELL CYCLE; CELL DIFFERENTIATION; CONTROLLED STUDY; MALIGNANT TRANSFORMATION; MOUSE; NONHUMAN; ONCOGENE; PREGNANCY; PRIORITY JOURNAL; TUMOR SUPPRESSOR GENE;

ANIMALS; ANTIGENS, POLYOMAVIRUS TRANSFORMING; CELL CYCLE; CELL DIFFERENTIATION; CROSSES, GENETIC; FEMALE; GENES, BRCA1; GENES, RETINOBLASTOMA; MAMMARY GLANDS, ANIMAL; MAMMARY NEOPLASMS, EXPERIMENTAL; MAMMARY TUMOR VIRUS, MOUSE; MICE; MICE, KNOCKOUT; MICE, TRANSGENIC; MILK PROTEINS; ONCOGENES; PREGNANCY; PROTO-ONCOGENE PROTEINS; RECEPTORS, CELL SURFACE; RECEPTORS, NOTCH; RETINOBLASTOMA PROTEIN; TRANSFORMING GROWTH FACTOR BETA; TRANSFORMING GROWTH FACTOR BETA1; TRANSGENES;

EID: 0035950529 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1204888 Document Type: Article

Times cited : (30)

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