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Volumn 57, Issue 9, 1997, Pages 1664-1667

Genetic interactions between atm and p53 influence cellular proliferation and irradiation-induced cell cycle checkpoints

Author keywords

[No Author keywords available]

Indexed keywords

ANIMAL CELL; ANIMAL MODEL; ARTICLE; ATAXIA TELANGIECTASIA; CELL CYCLE G1 PHASE; CELL CYCLE S PHASE; CELL PROLIFERATION; EMBRYO; EMBRYO CELL; FIBROBLAST CULTURE; HOMOZYGOSITY; IRRADIATION; MALIGNANT NEOPLASTIC DISEASE; MOUSE; NONHUMAN; PRIORITY JOURNAL;

EID: 0030941281     PISSN: 00085472     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article
Times cited : (74)

References (18)
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  • 7
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    • Hawley, R.S.1    Friend, S.H.2
  • 8
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    • Mice lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control
    • Deng, C., Zhang, P., Harper, J. W., Elledge, S. J., and Leder, P. Mice lacking p21CIP1/WAF1 undergo normal development, but are defective in G1 checkpoint control. Cell, 82: 675-684, 1995.
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    • High spontaneous intrachromosomal recombination rates in ataxiatelangiectasia
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    • Meyn, M.S.1
  • 10
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    • Meyn, M. S., Strasfeld, L., and Allen, C. Testing the role of p53 in the expression of genetic instability and apoptosis in ataxia-telangiectasia. Int. J. Radiat. Biol., 66: S141-S149, 1994.
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  • 13
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* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.