Wild-type and mutated nicastrins do not display aminopeptidase M- and B-like activities

Biochemical and Biophysical Research Communications

Volumn 289, Issue 3, 2001, Pages 678-680

  Fergani, Anissa ^a   Yu, G ^b   St George Hyslop, Peter ^b   Checler, Frédéric ^a  

^a INSTITUT DE PHARMACOLOGIE MOLÉCULAIRE ET CELLULAIRE   (France)

^b UNIVERSITY OF TORONTO   (Canada)

Author keywords

Alzheimer's disease; Aminopeptidases; Mutant proteins; Nicastrin; Presenilins; Transfectants

Indexed keywords

AMINOPEPTIDASE B; AMYLOID BETA PROTEIN; AMYLOID PRECURSOR PROTEIN; MICROSOMAL AMINOPEPTIDASE; MUTANT PROTEIN; NICASTRIN; PRESENILIN 1; PRESENILIN 2; PROTEIN;

ALZHEIMER DISEASE; ARTICLE; CATALYSIS; CONTROLLED STUDY; EMBRYO; ENZYME ACTIVITY; FAMILIAL DISEASE; GENE DELETION; GENE MUTATION; GENETIC TRANSFECTION; HUMAN; HUMAN CELL; HYDROPHILICITY; MISSENSE MUTATION; PRIORITY JOURNAL; PROTEIN PROTEIN INTERACTION; PROTEIN SYNTHESIS; SEQUENCE HOMOLOGY;

EID: 0035824426     PISSN: 0006291X     EISSN: None     Source Type: Journal    
DOI: 10.1006/bbrc.2001.6030     Document Type: Article

References (23)