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Volumn 167, Issue 7, 2001, Pages 3725-3733

IL-4 therapy prevents the development of proteinuria in active heymann nephritis by inhibition of Tc1 cells

Author keywords

[No Author keywords available]

Indexed keywords

IMMUNOGLOBULIN G1; INTERLEUKIN 4; INTERLEUKIN 4 ANTIBODY; KIDNEY ANTIGEN; MONOCLONAL ANTIBODY; MONOCLONAL ANTIBODY MRCOX 81; PROTEIN CRRY; RECOMBINANT INTERLEUKIN 3; RECOMBINANT INTERLEUKIN 4; REGULATOR PROTEIN; UNCLASSIFIED DRUG;

EID: 0035478686     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: 10.4049/jimmunol.167.7.3725     Document Type: Article
Times cited : (29)

References (66)
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    • Aversa, G.1    Waugh, J.A.2    Hall, B.M.3
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    • Murine interstitial nephritis. III. The selection of phenotypic (Lyt and L3T4) and idiotypic (RE-Id) T cell preferences by genes in lgh-I and H-2K characterizes the cell-mediated potential for disease expression: Susceptible mice provide a unique effector T cell repertoire in response to tubular antigen
    • (1985) J. Immunol. , vol.134 , pp. 2375
    • Neilson, E.G.1    McCafferty, E.2    Mann, R.3    Michaud, L.4    Clayman, M.5
  • 65
    • 85037391148 scopus 로고    scopus 로고
    • Th2 immune deviation prevents active Heymann nephritis: The role of Th1 cytotoxic cells and macrophages: The 36th Australian and New Zealand Society of Nephrology annual meeting
    • Melbourne, Australia, March 2000
    • (2000)
    • Spicer, S.T.1    Boyd, R.2    He, C.3    Penny, M.4    Hall, B.M.5


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.