GDNF protects against aluminum-induced apoptosis in rabbits by upregulating Bcl-2 and Bcl-XL and inhibiting mitochondrial Bax translocation

Neurobiology of Disease

Volumn 8, Issue 5, 2001, Pages 764-773

  Ghribi, Othman ^a   Herman, Mary M ^b   Forbes, Michael S ^a   DeWitt, David A ^c   Savory, John ^a  

^a UNIVERSITY OF VIRGINIA   (United States)

^b NATIONAL INSTITUTE OF MENTAL HEALTH   (United States)

^c LIBERTY UNIVERSITY   (United States)

Author keywords

Aluminum; Bax; Bcl 2; Caspase 3; Cytochrome c; Endoplasmic reticulum; GDNF

Indexed keywords

ALUMINUM; ALUMINUM MALTOL; CASPASE 3; CYTOCHROME C; DNA; GLIAL CELL LINE DERIVED NEUROTROPHIC FACTOR; NEUROTOXIN; PROTEIN BAX; PROTEIN BCL 2; PROTEIN BCL X;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CONTROLLED STUDY; DEGENERATIVE DISEASE; ENDOPLASMIC RETICULUM; ENZYME ACTIVITY; FEMALE; IMMUNOHISTOCHEMISTRY; MITOCHONDRION; NERVE CELL NECROSIS; NEUROLOGIC DISEASE; NONHUMAN; PRIORITY JOURNAL; RABBIT;

EID: 0035173536     PISSN: 09699961     EISSN: None     Source Type: Journal    
DOI: 10.1006/nbdi.2001.0429     Document Type: Article

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