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Volumn 21, Issue 5, 2001, Pages 1444-1452
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Targeted genomic disruption of H-ras and N-ras, individually or in combination, reveals the dispensability of both loci for mouse growth and development
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Author keywords
[No Author keywords available]
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Indexed keywords
ANIMAL CELL;
ARTICLE;
CELL GROWTH;
CELL VIABILITY;
DNA TRANSCRIPTION;
EMBRYO DEVELOPMENT;
GENE DISRUPTION;
GENE FUNCTION;
GENE LOCATION;
GENE MUTATION;
GENE TARGETING;
MOUSE;
NONHUMAN;
ONCOGENE H RAS;
ONCOGENE K RAS;
ONCOGENE N RAS;
PRIORITY JOURNAL;
SEXUAL MATURATION;
ANIMALS;
BLOTTING, WESTERN;
BRAIN;
CELL DIFFERENTIATION;
CELL SEPARATION;
CELLS, CULTURED;
CROSSES, GENETIC;
EMBRYO;
FEMALE;
FERTILITY;
FLOW CYTOMETRY;
GENES, RAS;
GENOTYPE;
HETEROZYGOTE;
HIPPOCAMPUS;
LYMPHOCYTES;
MALE;
MICE;
MICE, KNOCKOUT;
MICROSCOPY, FLUORESCENCE;
MODELS, GENETIC;
MUTAGENESIS, SITE-DIRECTED;
NEURONS;
PHENOTYPE;
POLYMERASE CHAIN REACTION;
RAS PROTEINS;
REVERSE TRANSCRIPTASE POLYMERASE CHAIN REACTION;
SPLEEN;
STEM CELLS;
THYMUS GLAND;
ANIMALIA;
MAMMALIA;
RODENTIA;
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EID: 0035136213
PISSN: 02707306
EISSN: None
Source Type: Journal
DOI: 10.1128/MCB.21.5.1444-1452.2001 Document Type: Article |
Times cited : (259)
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References (50)
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