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Volumn 96, Issue 2, 2001, Pages 136-142

ERK and p38 MAP kinase activation are components of opioid-induced delayed cardioprotection

Author keywords

Delayed cardioprotection; ERK; Opioid; p38; Preconditioning

Indexed keywords

1,2,3,4,4A,5,12,12AALPHA OCTAHYDRO 4AALPHA (3 HYDROXYPHENYL) 2 METHYLQUINOLINO[2,3 G]ISOQUINOLINE; 2 (2 AMINO 3 METHOXYPHENYL)CHROMONE; 4 (4 FLUOROPHENYL) 2 (4 METHYLSULFINYLPHENYL) 5 (4 PYRIDYL)IMIDAZOLE; MITOGEN ACTIVATED PROTEIN KINASE; OPIATE; SYNAPTOPHYSIN;

EID: 0035074421     PISSN: 03008428     EISSN: None     Source Type: Journal    
DOI: 10.1007/s003950170063     Document Type: Article
Times cited : (102)

References (47)
  • 12
    • 0032503045 scopus 로고    scopus 로고
    • The p38-MAPK inhibitor, SB 203580, inhibits cardiac stress-activated protein kinases/c-Jun N-terminal kinases (SAPKs/JNKs)
    • (1998) FEBS Letters , vol.426 , pp. 93-96
    • Clerk, A.1    Sugden, P.2
  • 33
  • 41
    • 0030747146 scopus 로고    scopus 로고
    • Regulation of the ERK subgroup of MAP kinase cascades through G protein-coupled receptors
    • (1997) Cell Signal , vol.9 , pp. 337-351
    • Sugden, P.1    Clerk, A.2


* 이 정보는 Elsevier사의 SCOPUS DB에서 KISTI가 분석하여 추출한 것입니다.