KATP channels mediate late preconditioning against infarction produced by monophosphoryl lipid A

American Journal of Physiology

Volumn 271, Issue 6 PART 2, 1996, Pages

  Mei, David A ^a   Elliott, Gary T ^a   Gross, Garrett J ^b  

^a MEDICAL COLLEGE OF WISCONSIN   (United States)

^b Ribi ImmunoChem Research   (United States)

Author keywords

5 hydroxydecanoate; Action potential duration; Glibenclamide; Second window of protection

Indexed keywords

PHOSPHORYL LIPID A; POTASSIUM CHANNEL; ADENOSINE TRIPHOSPHATE; DRUG DERIVATIVE; LIPID A;

ANIMAL EXPERIMENT; ARTICLE; CORONARY ARTERY OBSTRUCTION; DOG; HEART INFARCTION SIZE; HEART PROTECTION; LEFT ANTERIOR DESCENDING CORONARY ARTERY; NONHUMAN; POTASSIUM CURRENT; PRIORITY JOURNAL; ACTION POTENTIAL; ANIMAL; COLLATERAL CIRCULATION; CORONARY ARTERY BLOOD FLOW; DRUG EFFECT; HEART INFARCTION; HEART INFARCTION PREVENTION; HEART MUSCLE; HEMODYNAMICS; PATHOLOGY; PATHOPHYSIOLOGY; PHYSIOLOGY; REACTION TIME;

ACTION POTENTIALS; ADENOSINE TRIPHOSPHATE; ANIMALS; COLLATERAL CIRCULATION; CORONARY CIRCULATION; DOGS; HEMODYNAMIC PROCESSES; ISCHEMIC PRECONDITIONING, MYOCARDIAL; LIPID A; MYOCARDIAL INFARCTION; MYOCARDIUM; POTASSIUM CHANNELS; REACTION TIME;

EID: 0030459703     PISSN: 00029513     EISSN: None     Source Type: Journal    
DOI: 10.1152/ajpheart.1996.271.6.h2723     Document Type: Article

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