Biology of human papillomaviruses

International Journal of Experimental Pathology

Volumn 82, Issue 1, 2001, Pages 15-33

  Mcmurray, H R ^a   Nguyen, D ^a   Westbrook, T F ^a   Mcance, D J ^a  

^a UNIVERSITY OF ROCHESTER MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

VIRUS PROTEIN;

ARTICLE; CELL CYCLE G1 PHASE; CELL CYCLE S PHASE; GENE EXPRESSION; GENITAL TRACT CANCER; KERATINOCYTE; NONHUMAN; PRIORITY JOURNAL; SQUAMOUS CELL CARCINOMA; VIRUS GENOME; VIRUS PARTICLE; VIRUS REPLICATION; WART VIRUS;

ADENOSINE TRIPHOSPHATASES; ADULT; APOPTOSIS; CARCINOMA, SQUAMOUS CELL; CELL CYCLE; CELL DEATH; CELL TRANSFORMATION, VIRAL; CYCLINS; EPITHELIUM; FEMALE; GENITAL NEOPLASMS, FEMALE; GENITAL NEOPLASMS, MALE; HISTONE DEACETYLASES; HUMANS; KERATINOCYTES; MALE; MIDDLE AGED; ONCOGENE PROTEINS, VIRAL; PAPILLOMAVIRIDAE; PROTEIN BINDING; RECEPTORS, GROWTH FACTOR; REPRESSOR PROTEINS; RETINOBLASTOMA PROTEIN; TRANSCRIPTION FACTOR AP-1; TRANSCRIPTION, GENETIC; VIRAL ENVELOPE PROTEINS; VIRUS REPLICATION;

EID: 0034955648     PISSN: 09599673     EISSN: None     Source Type: Journal    
DOI: 10.1046/j.1365-2613.2001.00177.x     Document Type: Article

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59. Two distinct and frequently mutated regions of retinoblastoma protein are required for binding to SV40 T antigen
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61. A cellular protein mediates association of p53 with the E6 oncoprotein of human papillomavirus types 16 or 18
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99. Rb binds c-Jun and activates transcription
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102. Altered cell cycle regulation in the lens of HPV-16 E6 or E7 transgenic mice: Implications for tumor suppressor gene function in development
103. Temporally distinct patterns of p53-dependent and p53-independent apoptosis during mouse lens development
104. The G (2) checkpoint is maintained by redundant pathways
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106. Identification of a novel retinoblastoma gene product binding site on human papillomavirus type 16, E7 protein
107. Activation of the platelet-derived growth factor receptor by the bovine papillomavirus E5 transforming protein
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110. Human papillomavirus type 16, E5 gene stimulates the transforming activity of the epidermal growth factor receptor
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114. Single amino acid substitutions in 'low-risk' human papillomavirus (HPV) type 6, E7 protein enhance features characteristic of the 'high-risk' HPV E7 oncoproteins
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117. The epidemiology of cervical carcinogenesis
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119. The papillomavirus E1 protein forms a DNA-dependent hexameric complex with ATPase and DNA helicase activities
120. High-intensity Raf signal causes cell cycle arrest mediated by p21Cip1
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125. Bovine papillomavirus transcriptional regulation: Localization of the E2-responsive elements of the long control region
126. E5 oncoprotein retained in the endoplasmic reticulum/cis Golgi still induces PDGF receptor autophosphorylation but does not transform cells
127. The human papillomavirus type 16, E6 and E7 oncoproteins dissociate cellular telomerase activity from the maintenance of telomere length
128. The E5 gene of HPV-16 enhances keratinocyte immortalization by full-length DNA
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130. The E5 oncoprotein of human papillomavirus type 16 inhibits the acidification of endosomes in human keratinocytes
131. The E5 oncoprotein of human papillomavirus type 16 transforms fibroblasts and effects the downregulation of the epidermal growth factor receptor in keratinocytes
132. Mapping of HPV-11 E1 binding site and determination of other important cis elements for replication of the origin
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134. Determination of the functional difference between human papillomavirus type 6 and 16, E7 proteins by their 30 N-terminal amino acid residues
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136. Inhibition of Bak-induced apoptosis by HPV-18 KG
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138. Transient replication of BPV-1 requires two viral polypeptides encoded by the E1 and E2 open reading frames
139. The human papillomavirus HPV-6 and HPV-16 E5 proteins co-operate with HPV-16 E7 in the transformation of primary rodent cells
140. The human papillomavirus (HPV) 16, E2 protein induces apoptosis in the absence of other HPV proteins and via a p53-dependent pathway
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142. The human papillomavirus E7 oncoprotein and the cellular transcription factor E2F bind to separate sites on the retinoblastoma tumor suppressor protein
143. Sequential activation of cyclin E and cyclin A gene expression by human papillomavirus type 16, E7 through sequences necessary for transformation
144. The human papillomavirus type 16, E6 oncoprotein can down-regulate p53 activity by targeting the transcriptional coactivator CBP/p300
145. Viruses in human cancers