Investigation of elements sufficient to imprint the mouse Air promoter

Molecular and Cellular Biology

Volumn 21, Issue 15, 2001, Pages 5008-5017

  Sleutels, F ^a   Barlow, D P ^a  

^a ERICH SCHMID INSTITUTE OF MATERIALS SCIENCE   (Austria)

Author keywords

[No Author keywords available]

Indexed keywords

ADENINE PHOSPHORIBOSYLTRANSFERASE; COMPLEMENTARY RNA; SOMATOMEDIN B RECEPTOR;

ALLELE; ARTICLE; CPG ISLAND; DNA METHYLATION; GENOME IMPRINTING; INTRON; MOUSE; NONHUMAN; NUCLEOTIDE SEQUENCE; PRIORITY JOURNAL; PROMOTER REGION; TRANSGENE;

ADENINE PHOSPHORIBOSYLTRANSFERASE; ALLELES; ANIMALS; CPG ISLANDS; DNA; DNA METHYLATION; FEMALE; GENOMIC IMPRINTING; INTRONS; MALE; MICE; MICE, TRANSGENIC; MODELS, GENETIC; MOTHERS; OLIGONUCLEOTIDES, ANTISENSE; PHENOTYPE; PROMOTER REGIONS (GENETICS); RNA; TRANSGENES;

EID: 0034947995     PISSN: 02707306     EISSN: None     Source Type: Journal    
DOI: 10.1128/MCB.21.15.5008-5017.2001     Document Type: Article

References (40)

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2. Cre-mediated somatic site-specific recombination in mice
3. CpG islands as genomic footprints of promoters that are associated with replication origins
4. Competition - A common motif for the imprinting mechanism
5. Methylation and imprinting: From host defense to gene regulation
6. Insulators and boundaries: Versatile regulatory elements in the eukaryotic genome
7. Imprinting: Focusing on the center
8. De novo deletions of SNRPN exon 1 in early human and mouse embryos result in a paternal to maternal imprint switch
9. The imprinting box of the mouse Igf2r gene
10. Analysis of murine Snrpn and human SNRPN gene imprinting in transgenic mice
11. Sp1 elements protect a CpG island from de novo methylation
12. A silencer element identified in Drosophila is required for imprinting of H19 reporter transgenes in mice
13. Multiple mechanisms regulate imprinting of the mouse distal chromosome 7 gene cluster
14. A 5′ differentially methylated sequence and the 3′-flanking region are necessary for H19 transgene imprinting
15. A transcriptional insulator at the imprinted H19/Igf2 locus
16. Role for DNA methylation in genomic imprinting
17. The imprinted antisense RNA at the Igf2r locus overlaps but does not imprint Mas1
18. Sp1 sites in the mouse aprt gene promoter are required to prevent methylation of the CpG island
19. Epigenetic gene inactivation induced by a cis-acting methylation center
20. Characteristics of imprinted genes
21. DNA methylation and chromatin modification
22. Sequence and comparative analysis of the mouse 1-megabase region orthologous to the human 11p15 imprinted domain
23. The structural H19 gene is required for transgene imprinting
24. Regulation of chromatin structure by site-specific histone H3 methyltransferases
25. Enhancer competition between H19 and Igf2 does not mediate their imprinting
26. Structure of the imprinted mouse Snrpn gene and establishment of its parental-specific methylation pattern
27. The imprinting box of the Prader-Willi/Angelman syndrome domain
28. The uniqueness of the imprinting mechanism
29. H19 and Igf2 monoallelic expression is regulated in two distinct ways by a shared cis acting regulatory region upstream of H19
30. Role of a 461-bp G-rich repetitive element in H19 transgene imprinting
31. Maternal-specific methylation of the imprinted mouse Igf2r locus identifies the expressed locus as carrying the imprinting signal
32. Transgenes as molecular probes for genomic imprinting
33. The mouse insulin-like growth factor II/cation-independent mannose 6-phosphate (IGF-II/MPR) receptor gene: Molecular cloning and genomic organization
34. Deletion of the H19 differentially methylated domain results in loss of imprinted expression of H19 and Igf2
35. The sins of the fathers and mothers: Genomic imprinting in mammalian development
36. A paternal-specific methylation imprint marks the alleles of the mouse H19 gene
37. Determination of spontaneous loss of heterozygosity mutations in Aprt heterozygous mice
38. Cytosine methylation and mammalian development
39. Imprinted expression of the Igf2r gene depends on an intronic CpG island
40. Non-imprinted Igf2r expression decreases growth and rescues the Tme mutation in mice