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Volumn 170, Issue 1, 2001, Pages 207-215
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Long-term thyroxine administration increases heat stress protein-70 mRNA expression and attenuates p38 MAP kinase activity in response to ischaemia
a a a a a a a a |
Author keywords
[No Author keywords available]
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Indexed keywords
HEAT SHOCK PROTEIN 70;
LEVOTHYROXINE;
MESSENGER RNA;
MITOGEN ACTIVATED PROTEIN KINASE;
THYROXINE;
ABDOMINAL AORTA;
ANIMAL EXPERIMENT;
ANIMAL MODEL;
ANIMAL TISSUE;
AORTA CONSTRICTION;
ARTICLE;
CONTROLLED STUDY;
ENZYME ACTIVITY;
ENZYME PHOSPHORYLATION;
GENE EXPRESSION;
GENE INDUCTION;
HEART;
HEART HYPERTROPHY;
HEART MUSCLE ISCHEMIA;
HYPERTHYROIDISM;
ISOLATED HEART;
MALE;
NONHUMAN;
PERFUSION;
PRIORITY JOURNAL;
PROTEIN EXPRESSION;
RAT;
RAT STRAIN;
REPERFUSION;
THYROID HORMONE BLOOD LEVEL;
TIME;
ANIMALS;
CARDIOMEGALY;
CHRONIC DISEASE;
ELECTROPHORESIS, POLYACRYLAMIDE GEL;
HSP70 HEAT-SHOCK PROTEINS;
HYPERTHYROIDISM;
IMMUNOBLOTTING;
MALE;
MITOGEN-ACTIVATED PROTEIN KINASES;
MYOCARDIAL ISCHEMIA;
MYOCARDIUM;
P38 MITOGEN-ACTIVATED PROTEIN KINASES;
PERFUSION;
PHOSPHORYLATION;
PROTEIN KINASE C;
RATS;
RATS, WISTAR;
RNA, MESSENGER;
STATISTICS, NONPARAMETRIC;
THYROXINE;
TRIIODOTHYRONINE;
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EID: 0034913407
PISSN: 00220795
EISSN: None
Source Type: Journal
DOI: 10.1677/joe.0.1700207 Document Type: Article |
Times cited : (44)
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References (36)
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