Protein kinase B/Akt activates c-Jun NH2-terminal kinase by increasing NO production in response to shear stress

Journal of Applied Physiology

Volumn 91, Issue 4, 2001, Pages 1574-1581

  Go, Young Mi ^a   Boo, Yong Chool ^a   Park, Heonyong ^a   Maland, Matthew C ^a   Patel, Rakesh ^b   Pritchard Jr , Kirkwood A ^c   Fujio, Yasushi ^d   Walsh, Kenneth ^d   Darley Usmar, Victor ^b   Jo, Hanjoong ^a,e  

^a Emory University   (United States)

^b University of Alabama at Birmingham   (United States)

^c MEDICAL COLLEGE OF WISCONSIN   (United States)

^d TUFTS UNIVERSITY SCHOOL OF MEDICINE   (United States)

^e EMORY UNIVERSITY   (United States)

Author keywords

Atherosclerosis; Endothelial cells; Endothelial nitric oxide synthase; Mechanosensing; Mitogen activated protein kinase

Indexed keywords

MITOGEN ACTIVATED PROTEIN KINASE; NITRIC OXIDE; NITRIC OXIDE SYNTHASE; OXYGEN; PHOSPHATIDYLINOSITOL 3 KINASE; PHOSPHATIDYLINOSITOL 3 KINASE INHIBITOR; PROTEIN KINASE B; RAS PROTEIN; STRESS ACTIVATED PROTEIN KINASE; VIRUS VECTOR; WORTMANNIN;

ADENOVIRUS; ANIMAL CELL; AORTA; ARTICLE; ATHEROGENESIS; CATTLE; CONTROLLED STUDY; ENZYME ACTIVATION; NONHUMAN; PRIORITY JOURNAL; PROTEIN EXPRESSION; SHEAR STRESS; VASCULAR ENDOTHELIUM;

EID: 0034813334     PISSN: 87507587     EISSN: None     Source Type: Journal    
DOI: 10.1152/jappl.2001.91.4.1574     Document Type: Article

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