Catecholamines potentiate amyloid β-peptide neurotoxicity: Involvement of oxidative stress, mitochondrial dysfunction, and perturbed calcium homeostasis

Neurobiology of Disease

Volumn 5, Issue 4, 1998, Pages 229-243

  Fu, Weiming ^a   Luo, Hong ^a   Parthasarathy, Sampath ^b   Mattson, Mark P ^a  

^a UNIVERSITY OF KENTUCKY   (United States)

^b EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

Alzheimer's disease; Apoptosis; Dopamine; Epinephrine; Hippocampus; Lipid peroxidation; Norepinephrine

Indexed keywords

ACETYLCHOLINE; ADRENALIN; ALPHA TOCOPHEROL; CALCIUM; CATECHOLAMINE; DOPAMINE; DOPAMINE RECEPTOR; GALLIC ACID PROPYL ESTER; GLUTATHIONE; NORADRENALIN; PROPRANOLOL DERIVATIVE; SEROTONIN;

ALZHEIMER DISEASE; ANIMAL CELL; ARTICLE; CALCIUM HOMEOSTASIS; CELL CULTURE; CELL DEATH; CELL SURVIVAL; DEGENERATIVE DISEASE; EMBRYO; HIPPOCAMPUS; MEMBRANE POTENTIAL; NONHUMAN; OXIDATIVE STRESS; PRIORITY JOURNAL; RAT; STATISTICAL ANALYSIS;

EID: 0031773366     PISSN: 09699961     EISSN: None     Source Type: Journal    
DOI: 10.1006/nbdi.1998.0192     Document Type: Article

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