β-amyloid and ionophore A23187 evoke tau hyperphosphorylation by distinct intracellular pathways: Differential involvement of the calpain/protein kinase C system

Journal of Neuroscience Research

Volumn 49, Issue 6, 1997, Pages 759-768

  Shea, Thomas B ^a,b   Prabhakar, Sathya ^a   Ekinci, Fatma J ^a  

^a University of Massachusetts Lowell   (United States)

^b UNIVERSITY OF MASSACHUSETTS LOWELL   (United States)

Author keywords

amyloid; Alzheimer's disease; Calcium; Calcium ionophore; Calpain; Paired helical filaments; PHF 1; Phosphorylation; Protein kinase C; Tau

Indexed keywords

1 (5 ISOQUINOLINESULFONYL) 2 METHYLPIPERAZINE; AMINO ACID; AMYLOID BETA PROTEIN; ANTIBODY; ANTISENSE OLIGONUCLEOTIDE; CALCIMYCIN; CALCIUM; CALPAIN; CALPEPTIN; EGTAZIC ACID; EPITOPE; PHOSPHATE; PROTEIN KINASE C; SYNTHETIC PEPTIDE; TAU PROTEIN;

ALZHEIMER DISEASE; ARTICLE; CALCIUM TRANSPORT; CONTROLLED STUDY; HUMAN; HUMAN CELL; IMMUNOBLOTTING; IMMUNOCYTOCHEMISTRY; PRIORITY JOURNAL; PROTEIN PHOSPHORYLATION;

AMYLOID BETA-PROTEIN; ANTIBODIES, MONOCLONAL; ANTIBODY SPECIFICITY; BIOLOGICAL MARKERS; CALCIMYCIN; CALCIUM; CALPAIN; EPITOPES; HUMANS; IONOPHORES; NEUROBLASTOMA; PHOSPHOPROTEINS; PHOSPHORYLATION; PROTEIN KINASE C; TAU PROTEINS; TUMOR CELLS, CULTURED;

EID: 0030986545     PISSN: 03604012     EISSN: None     Source Type: Journal    
DOI: 10.1002/(SICI)1097-4547(19970915)49:6<759::AID-JNR10>3.0.CO;2-N     Document Type: Article

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