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Volumn 275, Issue 30, 2000, Pages 23059-23064

A four amino acid deletion polymorphism in the third intracellular loop of the human α(2C)-adrenergic receptor confers impaired coupling to multiple effectors

(5) Small, Kersten M a Forbes, Susan L a Rahman, Fahema F a Bridges, Kari M a Liggett, Stephen B a

a UNIVERSITY OF CINCINNATI COLLEGE OF MEDICINE (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADENYLATE CYCLASE; ALPHA 2 ADRENERGIC RECEPTOR; AMINO ACID DERIVATIVE; GUANINE NUCLEOTIDE BINDING PROTEIN; MITOGEN ACTIVATED PROTEIN KINASE;

ANIMAL CELL; ARTICLE; CHROMOSOMAL LOCALIZATION; CHROMOSOME 4; GENE DELETION; GENE FREQUENCY; GENE FUNCTION; GENE LOCUS; GENE LOSS; GENE REPRESSION; GENETIC POLYMORPHISM; GENETIC VARIABILITY; NONHUMAN; NUCLEOTIDE SEQUENCE; PHENOTYPE; PRIORITY JOURNAL; PROTEIN DOMAIN;

ADRENERGIC ALPHA-AGONISTS; AMINO ACID SEQUENCE; AMINO ACIDS; ANIMALS; BASE SEQUENCE; CRICETINAE; DNA PRIMERS; HUMANS; MOLECULAR SEQUENCE DATA; POLYMORPHISM, GENETIC; PROTEIN BINDING; RECEPTORS, ADRENERGIC, ALPHA-2; SEQUENCE DELETION; SIGNAL TRANSDUCTION;

ANIMALIA; CRICETINAE; CRICETULUS GRISEUS;

EID: 0034725616 PISSN: 00219258 EISSN: None Source Type: Journal
DOI: 10.1074/jbc.M000796200 Document Type: Article

Times cited : (174)

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