Glutamate transporter EAAT2 splice variants occur not only in ALS, but also in AD and controls

Neurology

Volumn 55, Issue 8, 2000, Pages 1082-1088

  Honig, L S ^a,c   Chambliss, D D ^a   Bigio, E H ^a   Carroll, S L ^b   Elliott, J L ^a  

^a UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

^b Baptist Health   (United States)

^c The New York Presbyterian Hospital   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

COMPLEMENTARY DNA; EXCITATORY AMINO ACID; GLUTAMIC ACID; MESSENGER RNA;

ALTERNATIVE RNA SPLICING; ALZHEIMER DISEASE; AMYOTROPHIC LATERAL SCLEROSIS; ARTICLE; CLINICAL ARTICLE; CONTROLLED STUDY; DEGENERATIVE DISEASE; DEMENTIA; HUMAN; HUMAN TISSUE; MOTOR NEURON DISEASE; NEUROLOGIC DISEASE; NEUROPATHOLOGY; POLYMERASE CHAIN REACTION; PRIORITY JOURNAL;

EID: 0034711151     PISSN: 00283878     EISSN: None     Source Type: Journal    
DOI: 10.1212/WNL.55.8.1082     Document Type: Article

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