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Volumn 19, Issue 36, 2000, Pages 4134-4145

Blockade of Smad4 in transformed keratinocytes containing a Ras oncogene leads to hyperactivation of the Ras-dependent Erk signalling pathway associated with progression to undifferentiated carcinomas

(4) Iglesias, Maite a Frontelo, Pilar a Gamallo, Carlos b Quintanilla, Miguel a

a INSTITUTO DE INVESTIGACIONES BIOMÉDICAS ALBERTO SOLS (Spain)

b HOSPITAL UNIVERSITARIO DE LA PRINCESA (Spain)

Author keywords

Carcinoma; Erk; Ras; Smad4; TGF 1; Urokinase

Indexed keywords

2 (2 AMINO 3 METHOXYPHENYL)CHROMONE; PROTEIN P21; RAS PROTEIN; SMAD PROTEIN; TRANSCRIPTION FACTOR; TRANSFORMING GROWTH FACTOR BETA; TRANSFORMING GROWTH FACTOR BETA1; UROKINASE;

ANIMAL CELL; ARTICLE; CANCER GROWTH; CELL GROWTH; CELL TRANSFORMATION; CONTROLLED STUDY; GENE ACTIVATION; GENE CONTROL; GENE FUNCTION; GENE INDUCTION; GENE LOSS; KERATINOCYTE; MOUSE; NONHUMAN; ONCOGENE RAS; PRIORITY JOURNAL; SIGNAL TRANSDUCTION; UNDIFFERENTIATED CARCINOMA;

EID: 0034710545 PISSN: 09509232 EISSN: None Source Type: Journal
DOI: 10.1038/sj.onc.1203764 Document Type: Article

Times cited : (63)

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