Effects of a K+ channel opener to reduce transmural dispersion of repolarization and prevent torsade de pointes in LQT1, LQT2, and LQT3 models of the Long-QT syndrome

Circulation

Volumn 102, Issue 6, 2000, Pages 706-712

  Shimizu, Wataru ^a   Antzelevitch, Charles ^a  

^a MASONIC MEDICAL RESEARCH LABORATORY   (United States)

Author keywords

Arrhythmia; Cells; Genes; Long QT syndrome; Nicorandil

Indexed keywords

NICORANDIL; POTASSIUM CHANNEL; POTASSIUM CHANNEL STIMULATING AGENT; POTASSIUM ION;

ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; DOG; DRUG MECHANISM; HEART MUSCLE POTENTIAL; HEART REPOLARIZATION; LONG QT SYNDROME; NONHUMAN; PRIORITY JOURNAL; TORSADE DES POINTES;

EID: 0034622604     PISSN: 00097322     EISSN: None     Source Type: Journal    
DOI: 10.1161/01.CIR.102.6.706     Document Type: Article

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