BCL-6-deficient mice reveal an IL-4-independent, STAT6-dependent pathway that controls susceptibility to infection by Leishmania major

Journal of Immunology

Volumn 163, Issue 4, 1999, Pages 2098-2103

  Dent, Alexander L ^a   Doherty, T Mark ^b   Paul, William E ^b   Sher, Alan ^b   Staudt, Louis M ^a  

^a NATIONAL CANCER INSTITUTE   (United States)

^b NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

CYTOKINE; GAMMA INTERFERON; INTERLEUKIN 13; INTERLEUKIN 4; STAT6 PROTEIN;

ANIMAL CELL; ANIMAL MODEL; ARTICLE; CONTROLLED STUDY; DISEASE PREDISPOSITION; IMMUNE RESPONSE; LEISHMANIA MAJOR; LEISHMANIASIS; LYMPHOCYTE DIFFERENTIATION; MOUSE; NONHUMAN; PARASITE; PRIORITY JOURNAL; T LYMPHOCYTE SUBPOPULATION;

ANIMALS; DISEASE SUSCEPTIBILITY; DNA-BINDING PROTEINS; INTERLEUKIN-13; INTERLEUKIN-4; LEISHMANIA MAJOR; LEISHMANIASIS, CUTANEOUS; MICE; MICE, INBRED BALB C; MICE, INBRED C57BL; MICE, KNOCKOUT; PROTO-ONCOGENE PROTEINS; PROTO-ONCOGENE PROTEINS C-BCL-6; SIGNAL TRANSDUCTION; STAT6 TRANSCRIPTION FACTOR; TH2 CELLS; TRANS-ACTIVATORS; TRANSCRIPTION FACTORS;

EID: 0033566955     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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