Autoimmunity without diabetes in transgenic mice expressing β cell- specific CD86, but not CD80: Parameters that trigger progression to diabetes

Journal of Immunology

Volumn 161, Issue 5, 1998, Pages 2128-2140

  Guerder, Sylvie ^a,b   Eynon, Elizabeth E ^a   Flavell, Richard A ^a  

^a YALE UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b INSERM   (France)

Author keywords

[No Author keywords available]

Indexed keywords

B7 ANTIGEN; BROXURIDINE; CD86 ANTIGEN; GAMMA INTERFERON; GLUCOSE; INTERLEUKIN 4; MAJOR HISTOCOMPATIBILITY ANTIGEN CLASS 2; T LYMPHOCYTE RECEPTOR; TUMOR NECROSIS FACTOR ALPHA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOIMMUNITY; CELL SPECIFICITY; CONTROLLED STUDY; DIABETES MELLITUS; LYMPHOCYTIC INFILTRATION; MOUSE; NONHUMAN; PANCREAS ISLET BETA CELL; PRIORITY JOURNAL; T LYMPHOCYTE ACTIVATION; TRANSGENIC MOUSE;

ANIMALS; ANTIGENS, CD; ANTIGENS, CD80; ANTIGENS, CD86; AUTOIMMUNITY; CELL MOVEMENT; CROSSES, GENETIC; DIABETES MELLITUS, EXPERIMENTAL; DISEASE PROGRESSION; HISTOCOMPATIBILITY ANTIGENS CLASS II; INSULIN; ISLETS OF LANGERHANS; LYMPHOCYTE ACTIVATION; MEMBRANE GLYCOPROTEINS; MICE; MICE, INBRED C57BL; MICE, INBRED CBA; MICE, TRANSGENIC; PROMOTER REGIONS (GENETICS); RATS; T-LYMPHOCYTES;

EID: 0032167602     PISSN: 00221767     EISSN: None     Source Type: Journal    
DOI: None     Document Type: Article

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