Glucocorticoid receptor promotes the function of myeloid-derived suppressor cells by suppressing HIF1α-dependent glycolysis

Cellular and Molecular Immunology

Volumn 15, Issue 6, 2018, Pages 618-629

  Lu, Yun ^a,b   Liu, Huanrong ^a,b   Bi, Yujing ^c   Yang, Hui ^a   Li, Yan ^a,b   Wang, Jian ^a,b   Zhang, Zhengguo ^a,b   Wang, Yu ^a,b   Li, Chunxiao ^a,b   Jia, Anna ^b   Han, Linian ^b   Hu, Ying ^b   Zhao, Yong ^d   Wang, Ruoning ^e   Liu, Guangwei ^a,b  

^a FUDAN UNIVERSITY   (China)

^b Beijing Normal University   (China)

^c BEIJING INSTITUTE OF MICROBIOLOGY AND EPIDEMIOLOGY   (China)

^d INSTITUTE OF ZOOLOGY   (China)

^e OHIO STATE UNIVERSITY   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

DEXAMETHASONE; GLUCOCORTICOID RECEPTOR; HEPATOCYTE NUCLEAR FACTOR 1ALPHA; NITRIC OXIDE; SHORT HAIRPIN RNA; HYPOXIA INDUCIBLE FACTOR 1ALPHA; LIPOPOLYSACCHARIDE; MIFEPRISTONE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BONE MARROW CELL; CELL FUNCTION; CELL METABOLISM; FLOW CYTOMETRY; GLYCOLYSIS; IMMUNOBLOTTING; IMMUNOLOGICAL TOLERANCE; INNATE IMMUNITY; LIVER INJURY; MOUSE; NONHUMAN; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN TARGETING; REGULATORY MECHANISM; REVERSE TRANSCRIPTION POLYMERASE CHAIN REACTION; SIGNAL TRANSDUCTION; SUPPRESSOR CELL; ANIMAL; C57BL MOUSE; DISEASE MODEL; DOWN REGULATION; DRUG EFFECT; HUMAN; INFLAMMATION; INJURY; LIVER; METABOLISM; MYELOID-DERIVED SUPPRESSOR CELL; PATHOLOGY;

ANIMALS; DEXAMETHASONE; DISEASE MODELS, ANIMAL; DOWN-REGULATION; GLYCOLYSIS; HUMANS; HYPOXIA-INDUCIBLE FACTOR 1, ALPHA SUBUNIT; IMMUNE TOLERANCE; IMMUNITY, INNATE; INFLAMMATION; LIPOPOLYSACCHARIDES; LIVER; MICE, INBRED C57BL; MIFEPRISTONE; MYELOID-DERIVED SUPPRESSOR CELLS; RECEPTORS, GLUCOCORTICOID; SIGNAL TRANSDUCTION;

EID: 85042910225     PISSN: 16727681     EISSN: None     Source Type: Journal    
DOI: 10.1038/cmi.2017.5     Document Type: Article

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