Hydrogen sulfide regulates cardiac mitochondrial biogenesis via the activation of AMPK

Journal of Molecular and Cellular Cardiology

Volumn 116, Issue , 2018, Pages 29-40

  Shimizu, Yuuki ^a   Polavarapu, Rohini ^a   Eskla, Kattri Liis ^a   Nicholson, Chad K ^a   Koczor, Christopher A ^a   Wang, Rui ^b   Lewis, William ^a   Shiva, Sruti ^c   Lefer, David J ^d   Calvert, John W ^a  

^a EMORY UNIVERSITY SCHOOL OF MEDICINE   (United States)

^b LAURENTIAN UNIVERSITY   (Canada)

^c UNIVERSITY OF PITTSBURGH SCHOOL OF MEDICINE   (United States)

^d LOUISIANA STATE UNIVERSITY SCHOOL OF MEDICINE   (United States)

Author keywords

AMPK; Heart; Hydrogen sulfide; Mitochondria

Indexed keywords

ADENOSINE TRIPHOSPHATE; CYSTATHIONINE GAMMA LYASE; HYDROGEN SULFIDE; HYDROXYMETHYLGLUTARYL COENZYME A REDUCTASE KINASE; PEROXISOME PROLIFERATOR ACTIVATED RECEPTOR GAMMA COACTIVATOR 1ALPHA; PRODRUG; SG 1002; UNCLASSIFIED DRUG; ENDOTHELIAL NITRIC OXIDE SYNTHASE; MITOCHONDRIAL DNA; NITRIC OXIDE; PHOSPHOPROTEIN PHOSPHATASE 2; PROTEIN SERINE THREONINE KINASE; STK11 PROTEIN, MOUSE; SULFUR;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; CELL NUCLEUS; CONTROLLED STUDY; ENZYME ACTIVATION; ENZYME DEFICIENCY; GENE OVEREXPRESSION; GENETIC ANALYSIS; GENETIC TRANSCRIPTION; HEART FAILURE; HEART FUNCTION; HEART MITOCHONDRION; HUMAN; HUMAN TISSUE; MITOCHONDRIAL BIOGENESIS; MITOCHONDRIAL RESPIRATION; MOUSE; NONHUMAN; PHARMACOLOGY; PRIORITY JOURNAL; PROTEIN FUNCTION; PROTEIN LOCALIZATION; REGULATORY MECHANISM; SUPPLEMENTATION; TRANSGENIC MOUSE; ANIMAL; C57BL MOUSE; DRUG EFFECT; GENETICS; METABOLISM; ORGANELLE BIOGENESIS; PHOSPHORYLATION;

AMP-ACTIVATED PROTEIN KINASES; ANIMALS; CELL NUCLEUS; DNA, MITOCHONDRIAL; ENZYME ACTIVATION; HEART FAILURE; HUMANS; HYDROGEN SULFIDE; MICE, INBRED C57BL; MITOCHONDRIA, HEART; NITRIC OXIDE; NITRIC OXIDE SYNTHASE TYPE III; ORGANELLE BIOGENESIS; PEROXISOME PROLIFERATOR-ACTIVATED RECEPTOR GAMMA COACTIVATOR 1-ALPHA; PHOSPHORYLATION; PROTEIN PHOSPHATASE 2; PROTEIN-SERINE-THREONINE KINASES; SULFUR;

EID: 85041406176     PISSN: 00222828     EISSN: 10958584     Source Type: Journal    
DOI: 10.1016/j.yjmcc.2018.01.011     Document Type: Article

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