Bypassing CFTR dysfunction in cystic fibrosis with alternative pathways for anion transport

Current Opinion in Pharmacology

Volumn 34, Issue , 2017, Pages 91-97

  Li, Hongyu ^a   Salomon, Johanna J ^b,c   Sheppard, David N ^a   Mall, Marcus A ^b,c   Galietta, Luis JV ^d  

^a UNIVERSITY OF BRISTOL   (United Kingdom)

^b UNIVERSITY HOSPITAL HEIDELBERG   (Germany)

^c UNIVERSITY OF HEIDELBERG   (Germany)

^d TELETHON INSTITUTE OF GENETICS AND MEDICINE TIGEM   (Italy)

Author keywords

[No Author keywords available]

Indexed keywords

CHLORIDE CHANNEL; CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR; PROTEIN SLC26A9; PROTEIN TMEM16A; SOLUTE CARRIER PROTEIN; UNCLASSIFIED DRUG; ANOCTAMIN 1; ANTIPORTER; SLC26A9 PROTEIN, HUMAN;

ANION TRANSPORT; CARBOXY TERMINAL SEQUENCE; CYSTIC FIBROSIS; HUMAN; NONHUMAN; PATHOPHYSIOLOGY; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN FUNCTION; PROTEIN LOCALIZATION; PROTEIN MOTIF; PROTEIN PHOSPHORYLATION; RESPIRATORY EPITHELIUM; REVIEW; ANIMAL; ION TRANSPORT; METABOLISM; RESPIRATORY MUCOSA;

ANIMALS; ANOCTAMIN-1; ANTIPORTERS; CYSTIC FIBROSIS; CYSTIC FIBROSIS TRANSMEMBRANE CONDUCTANCE REGULATOR; HUMANS; ION TRANSPORT; RESPIRATORY MUCOSA;

EID: 85032854316     PISSN: 14714892     EISSN: 14714973     Source Type: Journal    
DOI: 10.1016/j.coph.2017.10.002     Document Type: Review

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