TLR4 mutant mice are protected from renal fibrosis and chronic kidney disease progression

Physiological Reports

Volumn 3, Issue 9, 2015, Pages

  Souza, Ana C P ^a   Tsuji, Takayuki ^a,c   Baranova, Irina N ^b   Bocharov, Alexander V ^b   Wilkins, Kenneth J ^a   Street, Jonathan M ^a   Alvarez Prats, Alejandro ^a   Hu, Xuzhen ^a   Eggerman, Thomas ^a,b   Yuen, Peter S T ^a   Star, Robert A ^a  

^a NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES   (United States)

^b NATIONAL INSTITUTES OF HEALTH   (United States)

^c HAMAMATSU UNIVERSITY SCHOOL OF MEDICINE   (Japan)

Author keywords

Albuminuria; Endotoxemia; Inflammasome; Inflammation; Spleen apoptosis

Indexed keywords

CASPASE 3; CREATININE; CRYOPYRIN; HIGH MOBILITY GROUP B PROTEIN; PROGRAMMED DEATH 1 RECEPTOR; TOLL LIKE RECEPTOR 4;

ALBUMINURIA; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CHRONIC KIDNEY DISEASE; CONTROLLED STUDY; DISEASE COURSE; EPITHELIUM CELL; GLOMERULOSCLEROSIS; IMMUNOHISTOCHEMISTRY; KIDNEY FIBROSIS; MOUSE; NEPHRECTOMY; NONHUMAN; PROTEIN EXPRESSION; SPLEEN CELL; UPREGULATION; UREA NITROGEN BLOOD LEVEL;

EID: 85006043089     PISSN: None     EISSN: 2051817X     Source Type: Journal    
DOI: 10.14814/phy2.12558     Document Type: Article

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