M2A and M2C Macrophage Subsets Ameliorate Inflammation and Fibroproliferation in Acute Lung Injury Through Interleukin 10 Pathway

Shock

Volumn 48, Issue 1, 2017, Pages 119-129

  Tang, Lunxian ^a   Zhang, Hua ^a   Wang, Chunmei ^a   Li, Hongqiang ^a   Zhang, Qian ^a   Bai, Jianwen ^a  

^a TONGJI UNIVERSITY   (China)

Author keywords

Acute lung injury; fibrosis; IL 10; macrophage; neutrophil influx

Indexed keywords

ARGINASE 1; COLLAGEN TYPE 1; FIZZ1 PROTEIN; HYDROXYPROLINE; INTERLEUKIN 10; INTERLEUKIN 13; INTERLEUKIN 1BETA; INTERLEUKIN 4; JANUS KINASE 1; MANNOSE RECEPTOR; MYELOPEROXIDASE; PROTEIN; STAT3 PROTEIN; SUPPRESSOR OF CYTOKINE SIGNALING 3; TRANSFORMING GROWTH FACTOR BETA; TRANSFORMING GROWTH FACTOR BETA1; TUMOR NECROSIS FACTOR; UNCLASSIFIED DRUG; V SET DOMAIN CONTAINING T CELL ACTIVATION INHIBITOR 1;

ACUTE LUNG INJURY; ADOPTIVE TRANSFER; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; CELL COUNT; COMPARATIVE STUDY; CONTROLLED STUDY; CYTOKINE RELEASE; ENZYME ACTIVATION; ENZYME ACTIVITY; EX VIVO STUDY; FIBROSING ALVEOLITIS; GENE EXPRESSION; HISTOPATHOLOGY; IN VIVO STUDY; INFLAMMATION; LUNG FIBROSIS; LUNG PARENCHYMA; MACROPHAGE; MACROPHAGE MIGRATION; MALE; MOUSE; NONHUMAN; PATHOGENESIS; PHENOTYPE; PNEUMONIA; PROTEIN BLOOD LEVEL; PROTEIN EXPRESSION; ANIMAL; C57BL MOUSE; CELL DIFFERENTIATION; FIBROSIS; IMMUNOLOGY; METABOLISM; PHYSIOLOGY;

ACUTE LUNG INJURY; ANIMALS; APOPTOSIS; CELL DIFFERENTIATION; FIBROSIS; INFLAMMATION; INTERLEUKIN-10; MACROPHAGES; MALE; MICE; MICE, INBRED C57BL; STAT3 TRANSCRIPTION FACTOR;

EID: 85003881189     PISSN: 10732322     EISSN: 15400514     Source Type: Journal    
DOI: 10.1097/SHK.0000000000000820     Document Type: Article

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