TAT-SNAP-23 treatment inhibits the priming of neutrophil functions contributing to shock and/or sepsis-induced extra-pulmonary acute lung injury

Innate Immunity

Volumn 21, Issue 1, 2015, Pages 42-54

  Bai, Jianwen ^a   Tang, Lunxian ^a   Lomas Neira, Joanne ^b   Chen, Yaping ^b   McLeish, Kenneth R ^c,d   Uriarte, Silvia M ^c   Chung, Chun Shiang ^b   Ayala, Alfred ^b  

^a Key Laboratory of Arrhythmias of Ministry of Education   (China)

^b BROWN UNIVERSITY   (United States)

^c University of Louisville School of Medicine   (United States)

^d VETERANS AFFAIRS MEDICAL CENTER   (United States)

Author keywords

migration; Neutrophil influx; respiratory burst capacity

Indexed keywords

CXCL3 CHEMOKINE; ESTERASE; INTERLEUKIN 10; INTERLEUKIN 6; MONOCYTE CHEMOTACTIC PROTEIN 1; MYELOPEROXIDASE; SYNAPTOSOMAL ASSOCIATED PROTEIN 23; TRANSACTIVATOR PROTEIN; TUMOR NECROSIS FACTOR ALPHA; N ETHYLMALEIMIDE SENSITIVE FACTOR; PEROXIDASE; QB SNARE PROTEIN; QC SNARE PROTEIN; SNAP23 PROTEIN, MOUSE;

ACUTE LUNG INJURY; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ARTICLE; BLEEDING; BLOOD; CECAL LIGATION AND PUNCTURE; CELL MIGRATION; CONTROLLED STUDY; EXPERIMENTAL LIGATION; IN VITRO STUDY; IN VIVO STUDY; LUNG LAVAGE; LUNG PARENCHYMA; MALE; MOUSE; MOUSE MODEL; NEUTROPHIL; NEUTROPHIL CHEMOTAXIS; NONHUMAN; PROTEIN EXPRESSION; RESPIRATORY BURST; SEPSIS; SHOCK; ANIMAL; BRONCHOALVEOLAR LAVAGE FLUID; C57BL MOUSE; COMPLICATION; DRUG EFFECTS; IMMUNOLOGY; METABOLISM; PATHOPHYSIOLOGY; SEPTIC SHOCK;

ACUTE LUNG INJURY; ANIMALS; BRONCHOALVEOLAR LAVAGE FLUID; CHEMOTAXIS, LEUKOCYTE; GENE PRODUCTS, TAT; HEMORRHAGE; MICE; MICE, INBRED C57BL; N-ETHYLMALEIMIDE-SENSITIVE PROTEINS; NEUTROPHILS; PEROXIDASE; QB-SNARE PROTEINS; QC-SNARE PROTEINS; RESPIRATORY BURST; SEPSIS; SHOCK, SEPTIC;

EID: 84914141390     PISSN: 17534259     EISSN: 17534267     Source Type: Journal    
DOI: 10.1177/1753425913516524     Document Type: Article

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