Neuropathophysiology of Brain Injury

Anesthesiology Clinics

Volumn 34, Issue 3, 2016, Pages 453-464

  Quillinan, Nidia ^a   Herson, Paco S ^a   Traystman, Richard J ^a  

^a UNIVERSITY OF COLORADO SCHOOL OF MEDICINE   (United States)

Author keywords

Apoptosis; Excitotoxicity; Mechanisms of ischemic brain injury; Neuroinflammation; Neurovascular unit; Oxidative stress; Synaptic plasticity; Translation

Indexed keywords

ACIDOTOXICITY; APOPTOSIS; BRAIN INJURY; BRAIN ISCHEMIA; CELL INTERACTION; CELL REGENERATION; COMORBIDITY; ENCEPHALITIS; EXCITATION INHIBITION IMBALANCE; EXCITOTOXICITY; HUMAN; NERVE CELL PLASTICITY; NEUROLOGIC DISEASE; NEUROPATHOPHYSIOLOGY; NEUROPROTECTION; NONHUMAN; OXIDATIVE STRESS; PATHOPHYSIOLOGY; REVIEW; RISK FACTOR; TOXICITY; ANIMAL; CELL COMMUNICATION; COMPLICATION; INFLAMMATION; PHYSIOLOGY;

N METHYL DEXTRO ASPARTIC ACID RECEPTOR;

ANIMALS; APOPTOSIS; BRAIN INJURIES; CELL COMMUNICATION; HUMANS; INFLAMMATION; NEURONAL PLASTICITY; OXIDATIVE STRESS; RECEPTORS, N-METHYL-D-ASPARTATE;

EID: 84994438801     PISSN: 19322275     EISSN: 22103538     Source Type: Journal    
DOI: 10.1016/j.anclin.2016.04.011     Document Type: Review

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