Cell-extrinsic TNF collaborates with TRIF signaling to promote yersinia-induced apoptosis

Journal of Immunology

Volumn 197, Issue 10, 2016, Pages 4110-4117

  Peterson, Lance W ^a,b   Philip, Naomi H ^a,b   Dillon, Christopher P ^c   Bertin, John ^d   Gough, Peter J ^d   Green, Douglas R ^c   Brodsky, Igor E ^a,b  

^a UNIVERSITY OF PENNSYLVANIA   (United States)

^b UNIVERSITY OF PENNSYLVANIA   (United States)

^c ST JUDE CHILDREN S RESEARCH HOSPITAL   (United States)

^d GLAXOSMITHKLINE   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

BACTERIAL PROTEIN; CASPASE 3; CASPASE 8; IMMUNOGLOBULIN ENHANCER BINDING PROTEIN; INTERLEUKIN 1BETA CONVERTING ENZYME; MITOGEN ACTIVATED PROTEIN KINASE; PATTERN RECOGNITION RECEPTOR; RECEPTOR INTERACTING PROTEIN SERINE THREONINE KINASE; RECEPTOR INTERACTING PROTEIN SERINE THREONINE KINASE 1; TOLL LIKE RECEPTOR ADAPTOR MOLECULE 1; TUMOR NECROSIS FACTOR; UNCLASSIFIED DRUG; YOPJ PROTEIN; LACTATE DEHYDROGENASE; TICAM-1 PROTEIN, MOUSE; TOLL LIKE RECEPTOR 4; VESICULAR TRANSPORT ADAPTOR PROTEIN; YOPP PROTEIN, YERSINIA;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; BACTERIAL VIRULENCE; CONTROLLED STUDY; CYTOKINE PRODUCTION; CYTOKINE RESPONSE; ENZYME ACTIVITY; ENZYME INHIBITION; HOST RESISTANCE; MACROPHAGE; MOUSE; NONHUMAN; PRIORITY JOURNAL; PROTEIN CLEAVAGE; PROTEIN DEPLETION; PROTEIN EXPRESSION; PROTEIN FUNCTION; SIGNAL TRANSDUCTION; YERSINIA INFECTION; YERSINIA PSEUDOTUBERCULOSIS; ANIMAL; CELL DEATH; DEFICIENCY; GENETICS; IMMUNOLOGY; INNATE IMMUNITY; METABOLISM; MICROBIOLOGY; PATHOGENICITY; PHYSIOLOGY; PLASMID;

ADAPTOR PROTEINS, VESICULAR TRANSPORT; ANIMALS; APOPTOSIS; BACTERIAL PROTEINS; CASPASE 1; CELL DEATH; IMMUNITY, INNATE; L-LACTATE DEHYDROGENASE; MACROPHAGES; MICE; PLASMIDS; SIGNAL TRANSDUCTION; TOLL-LIKE RECEPTOR 4; TUMOR NECROSIS FACTORS; YERSINIA INFECTIONS; YERSINIA PSEUDOTUBERCULOSIS;

EID: 84994404364     PISSN: 00221767     EISSN: 15506606     Source Type: Journal    
DOI: 10.4049/jimmunol.1601294     Document Type: Article

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