Genetic ablation of calcium-independent phospholipase A2γ induces glomerular injury in mice

Journal of Biological Chemistry

Volumn 291, Issue 28, 2016, Pages 14468-14482

  Elimam, Hanan ^a,c   Papillon, Joan ^a   Kaufman, Daniel R ^a   Guillemette, Julie ^a   Aoudjit, Lamine ^a   Gross, Richard W ^b   Takano, Tomoko ^a,d   Cybulsky, Andrey V ^a,d  

^a RESEARCH INSTITUTE OF THE MCGILL UNIVERSITY HEALTH CENTRE   (Canada)

^b WASHINGTON UNIVERSITY SCHOOL OF MEDICINE   (United States)

^c Egypt   (Egypt)

^d Research Institute EM13244   (Canada)

Author keywords

[No Author keywords available]

Indexed keywords

CALCIUM; CELLS; CHEMICAL COMPOUNDS;

EPITHELIAL CELLS; GENETIC ABLATION; GLOMERULAR BASEMENT MEMBRANE; GLOMERULAR EPITHELIAL CELLS; PATHOPHYSIOLOGY; PERMSELECTIVITIES; PHOSPHOLIPASE A; WILD TYPES;

MAMMALS;

ALBUMIN; CALCIUM INDEPENDENT PHOSPHOLIPASE A2; MICROTUBULE ASSOCIATED PROTEIN; MICROTUBULE ASSOCIATED PROTEIN LIGHT CHAIN 3; NEPHRIN; SYNAPTOPODIN; UNCLASSIFIED DRUG; MEMBRANE PROTEIN; PHOSPHOLIPASE A2 GROUP VI; PLA2G6 PROTEIN, MOUSE;

AGING; ALBUMIN CREATININE RATIO; ALBUMINURIA; ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; AUTOPHAGY; CELL LOSS; CELL ULTRASTRUCTURE; CONTROLLED STUDY; DISORDERS OF MITOCHONDRIAL FUNCTIONS; DOWN REGULATION; ENZYME ACTIVITY; FEMALE; GENE DELETION; GENETIC ABLATION; GENETIC PROCEDURES; GLOMERULONEPHRITIS; GLOMERULUS; IPLA2GAMMA GENE; MALE; MOUSE; NONHUMAN; PODOCYTE; PRIORITY JOURNAL; PROTEIN EXPRESSION; PROTEIN URINE LEVEL; RENAL SYSTEM PARAMETERS; SEX DIFFERENCE; UNFOLDED PROTEIN RESPONSE; WILD TYPE; ANIMAL; C57BL MOUSE; CELL CULTURE; ENDOPLASMIC RETICULUM STRESS; GENETICS; KNOCKOUT MOUSE; METABOLISM; MITOCHONDRION; PATHOLOGY; PROTEINURIA;

AGING; ANIMALS; AUTOPHAGY; CELLS, CULTURED; ENDOPLASMIC RETICULUM STRESS; GLOMERULONEPHRITIS; GROUP VI PHOSPHOLIPASES A2; KIDNEY GLOMERULUS; MEMBRANE PROTEINS; MICE; MICE, INBRED C57BL; MICE, KNOCKOUT; MITOCHONDRIA; PHOSPHOLIPASES A2, CALCIUM-INDEPENDENT; PODOCYTES; PROTEINURIA;

EID: 84979018900     PISSN: 00219258     EISSN: 1083351X     Source Type: Journal    
DOI: 10.1074/jbc.M115.696781     Document Type: Article

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