Corrigendum: Endothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instability (Nature Communications (2016) 7 (11853) DOI: 10.1038/ncomms11853);Endothelial to mesenchymal transition is common in atherosclerotic lesions and is associated with plaque instability

Nature Communications

Volumn 7, Issue , 2016, Pages

  Evrard, Solene M ^a   Lecce, Laura ^a   Michelis, Katherine C ^a   Nomura Kitabayashi, Aya ^a   Pandey, Gaurav ^a   Purushothaman, K Raman ^a   D'Escamard, Valentina ^a   Li, Jennifer R ^a   Hadri, Lahouaria ^a   Fujitani, Kenji ^a   Moreno, Pedro R ^a   Benard, Ludovic ^a   Rimmele, Pauline ^a   Cohain, Ariella ^a   Mecham, Brigham ^b   Randolph, Gwendalyn J ^c   Nabel, Elizabeth G ^d   Hajjar, Roger ^a   Fuster, Valentin ^a,e   Boehm, Manfred ^f   Kovacic, Jason C ^a   more..

^a MOUNT SINAI SCHOOL OF MEDICINE   (United States)

^b Trialomics   (United States)

^c WASHINGTON UNIVERSITY   (United States)

^d BRIGHAM AND WOMEN S HOSPITAL   (United States)

^e CENTRO NACIONAL DE INVESTIGACIONES CARDIOVASCULARES CNIC   (Spain)

^f NATIONAL INSTITUTES OF HEALTH   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ALPHA SMOOTH MUSCLE ACTIN; CALVASCULIN; CD68 ANTIGEN; COLLAGEN; COLLAGEN 10A1; COLLAGEN 11A1; COLLAGEN 12A1; COLLAGEN 16A1; COLLAGEN 1A1; COLLAGEN 1A2; COLLAGEN 22A1; COLLAGEN 3A1; COLLAGEN 5A2; COLLAGEN 6A1; COLLAGEN 6A2; COLLAGEN 7A1; COLLAGEN 8A2; GELATINASE A; INTERSTITIAL COLLAGENASE; MATRILYSIN; MATRIX METALLOPROTEINASE 14; MYOSIN HEAVY CHAIN; SMAD3 PROTEIN; STROMELYSIN; STROMELYSIN 2; STROMELYSIN 3; TRANSCRIPTOME; TRANSFORMING GROWTH FACTOR BETA; UNCLASSIFIED DRUG; VASCULAR ENDOTHELIAL CADHERIN; VON WILLEBRAND FACTOR; BIOLOGICAL MARKER;

CARDIOVASCULAR DISEASE; CELLS AND CELL COMPONENTS; COLLAGEN; DETECTION METHOD; ENDOTHERMY; HEALTH CARE; MATRIX; OXIDATION; PATHOLOGY; PHENOTYPE;

ANIMAL CELL; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; APOPTOSIS; ARTICLE; ATHEROSCLEROSIS; ATHEROSCLEROTIC PLAQUE; BONE MARROW CELL; CELL HYPOXIA; CELL LINEAGE; CELL PROLIFERATION; CELL STRESS; CELL STRUCTURE; CELL TRANSFORMATION; CONTROLLED STUDY; ENDOTHELIAL MESENCHYMAL TRANSITION; ENDOTHELIUM CELL; FEMALE; FIBROBLAST; HUMAN; HUMAN CELL; HUMAN TISSUE; IMMUNOFLUORESCENCE; IN VITRO STUDY; LEUKOCYTE; MALE; MESENCHYME CELL; MONOCYTE; MOUSE; MYOFIBROBLAST; NONHUMAN; OXIDATIVE STRESS; PHENOTYPE; PROTEIN EXPRESSION; SIGNAL TRANSDUCTION; UBIQUITINATION; VASCULAR SMOOTH MUSCLE CELL; WESTERN BLOTTING; ANIMAL; CELL MOTION; EPITHELIAL MESENCHYMAL TRANSITION; METABOLISM; OXYGEN CONSUMPTION; PATHOLOGY; PHYSIOLOGY;

ANIMALS; ATHEROSCLEROSIS; BIOMARKERS; CELL LINEAGE; CELL MOVEMENT; CELL PROLIFERATION; ENDOTHELIAL CELLS; EPITHELIAL-MESENCHYMAL TRANSITION; HUMANS; MICE; OXIDATIVE STRESS; OXYGEN CONSUMPTION; PLAQUE, ATHEROSCLEROTIC; SIGNAL TRANSDUCTION; TRANSFORMING GROWTH FACTOR BETA;

EID: 84976349019     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms14710     Document Type: Erratum

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