Mechanisms of diabetes improvement following bariatric/metabolic surgery

Diabetes Care

Volumn 39, Issue 6, 2016, Pages 893-901

  Batterham, Rachel L ^a,b,c   Cummings, David E ^d  

^a UNIVERSITY COLLEGE LONDON   (United Kingdom)

^b UNIVERSITY COLLEGE LONDON HOSPITALS   (United Kingdom)

^c Camden and Islington National Health Service Foundation Trust   (United Kingdom)

^d UNIVERSITY OF WASHINGTON   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

FIBROBLAST GROWTH FACTOR 19; GLUCAGON LIKE PEPTIDE 1; INCRETIN; GASTROINTESTINAL HORMONE; GLUCOSE; GLUCOSE BLOOD LEVEL;

ARTICLE; BARIATRIC SURGERY; BILE ACID METABOLISM; CALORIC RESTRICTION; DIABETES MELLITUS; ENERGY METABOLISM; FOREGUT; GASTROINTESTINAL SURGERY; GLUCONEOGENESIS; GLUCOSE HOMEOSTASIS; GLUCOSE UTILIZATION; HUMAN; INSULIN SENSITIVITY; INTESTINE FLORA; METABOLIC REGULATION; NONHUMAN; PANCREAS ISLET BETA CELL; WEIGHT REDUCTION; BLOOD; DIABETES MELLITUS, TYPE 2; DIET THERAPY; GASTROINTESTINAL TRACT; GLUCOSE BLOOD LEVEL; INSULIN RESISTANCE; METABOLISM; OBESITY; PHYSIOLOGY; PROCEDURES; REMISSION; SIGNAL TRANSDUCTION;

BARIATRIC SURGERY; BLOOD GLUCOSE; CALORIC RESTRICTION; DIABETES MELLITUS, TYPE 2; ENERGY METABOLISM; GASTROINTESTINAL HORMONES; GASTROINTESTINAL TRACT; GLUCOSE; HUMANS; INCRETINS; INSULIN RESISTANCE; OBESITY; REMISSION INDUCTION; SIGNAL TRANSDUCTION; WEIGHT LOSS;

EID: 84971249928     PISSN: 01495992     EISSN: 19355548     Source Type: Journal    
DOI: 10.2337/dc16-0145     Document Type: Article

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