Deregulation of mitochondrial F1FO-ATP synthase via OSCP in Alzheimer's disease

Nature Communications

Volumn 7, Issue , 2016, Pages

  Beck, Simon J ^a   Guo, Lan ^a   Phensy, Aarron ^a   Tian, Jing ^a   Wang, Lu ^a,b   Tandon, Neha ^a   Gauba, Esha ^a   Lu, Lin ^a,b   Pascual, Juan M ^c   Kroener, Sven ^a   Du, Heng ^a,b  

^a UNIVERSITY OF TEXAS AT DALLAS   (United States)

^b SHANDONG UNIVERSITY   (China)

^c UNIVERSITY OF TEXAS SOUTHWESTERN MEDICAL CENTER   (United States)

Author keywords

[No Author keywords available]

Indexed keywords

ADENOSINE TRIPHOSPHATE; AMYLOID BETA PROTEIN[1-42]; BETA ACTIN; CYTOCHROME C OXIDASE; GLUTATHIONE TRANSFERASE; MITOCHONDRIAL ENZYME; OLIGOMYCIN SENSITIVITY CONFERRING PROTEIN; POSTSYNAPTIC DENSITY PROTEIN 95; PROTON TRANSPORTING ADENOSINE TRIPHOSPHATE SYNTHASE; REDUCED NICOTINAMIDE ADENINE DINUCLEOTIDE DEHYDROGENASE (UBIQUINONE); UNCLASSIFIED DRUG; VESICULAR GLUTAMATE TRANSPORTER 1; VOLTAGE DEPENDENT ANION CHANNEL; ADENOSINE TRIPHOSPHATASE; AMYLOID BETA PROTEIN; CARRIER PROTEIN; F1F0-ATP SYNTHASE; MEMBRANE PROTEIN; OLIGOMYCIN SENSITIVITY-CONFERRING PROTEIN;

AMINO ACID; BIOENERGETICS; BRAIN; DISEASE INCIDENCE; INJURY; MENTAL HEALTH; PERMEABILITY; PHYSIOLOGICAL RESPONSE; PROTEIN; RODENT;

ALZHEIMER DISEASE; ANIMAL EXPERIMENT; ANIMAL MODEL; ANIMAL TISSUE; ARTICLE; BRAIN MITOCHONDRION; BRAIN NERVE CELL; CONTROLLED STUDY; DISEASE COURSE; DOWN REGULATION; ENZYME REGULATION; EXCITATORY POSTSYNAPTIC POTENTIAL; FEMALE; HUMAN; HUMAN CELL; HUMAN TISSUE; LOSS OF FUNCTION MUTATION; MALE; MITOCHONDRIAL MEMBRANE POTENTIAL; MITOCHONDRIAL PERMEABILITY; MITOCHONDRIAL RESPIRATION; MITOCHONDRION SWELLING; MOUSE; NERVE CELL PLASTICITY; NONHUMAN; OXIDATIVE PHOSPHORYLATION; OXIDATIVE STRESS; PROTEIN EXPRESSION; PROTEIN PROTEIN INTERACTION; PROTEIN STABILITY; PROTEIN TARGETING; SYNAPSE VESICLE; SYNAPTIC TRANSMISSION; ANIMAL; BRAIN; DISEASE EXACERBATION; METABOLISM; MITOCHONDRION; NERVE CELL; TRANSGENIC MOUSE;

ADENOSINE TRIPHOSPHATASES; ADENOSINE TRIPHOSPHATE; ALZHEIMER DISEASE; AMYLOID BETA-PEPTIDES; ANIMALS; BRAIN; CARRIER PROTEINS; DISEASE PROGRESSION; HUMANS; MEMBRANE PROTEINS; MICE; MICE, TRANSGENIC; MITOCHONDRIA; MITOCHONDRIAL PROTON-TRANSLOCATING ATPASES; NEURONS; OXIDATIVE PHOSPHORYLATION; OXIDATIVE STRESS;

EID: 84967138102     PISSN: None     EISSN: 20411723     Source Type: Journal    
DOI: 10.1038/ncomms11483     Document Type: Article

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