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Volumn 351, Issue 6279, 2016, Pages 1333-1338
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Prostaglandin E2 constrains systemic inflammation through an innate lymphoid cell-IL-22 axis
a a a b a a b,i a c a d a a a e,f a a a g,h b,i more.. |
Author keywords
[No Author keywords available]
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Indexed keywords
INDOMETACIN;
INTERLEUKIN 22;
LIPOPOLYSACCHARIDE;
PROSTAGLANDIN E RECEPTOR 4;
PROSTAGLANDIN E2;
INTERLEUKIN DERIVATIVE;
INTERLEUKIN-22;
BACTERIUM;
CARDIOVASCULAR SYSTEM;
CELLS AND CELL COMPONENTS;
HOMEOSTASIS;
RISK FACTOR;
RODENT;
SIGNALING;
TRANSLOCATION;
ANIMAL CELL;
ANIMAL EXPERIMENT;
ARTICLE;
CELL DISRUPTION;
CONTROLLED STUDY;
CYTOKINE PRODUCTION;
DOWN REGULATION;
ENZYME INHIBITION;
GENE EXPRESSION;
HOMEOSTASIS;
INFLAMMATION;
INNATE IMMUNITY;
INTESTINE FLORA;
LYMPHOID CELL;
MOUSE;
NONHUMAN;
PRIORITY JOURNAL;
PROSTAGLANDIN SYNTHESIS;
PROTEIN EXPRESSION;
SIGNAL TRANSDUCTION;
ANIMAL;
ANTAGONISTS AND INHIBITORS;
BACTERIAL INFECTION;
GENETICS;
HUMAN;
IMMUNOLOGY;
INTESTINE;
LYMPHOCYTE;
MICROBIOLOGY;
MUS;
ANIMALS;
BACTERIAL INFECTIONS;
DINOPROSTONE;
GENE EXPRESSION;
HUMANS;
IMMUNITY, INNATE;
INFLAMMATION;
INTERLEUKINS;
INTESTINES;
LYMPHOCYTES;
MICE;
RECEPTORS, PROSTAGLANDIN E, EP4 SUBTYPE;
SIGNAL TRANSDUCTION;
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EID: 84961801657
PISSN: 00368075
EISSN: 10959203
Source Type: Journal
DOI: 10.1126/science.aad9903 Document Type: Article |
Times cited : (149)
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References (36)
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